Rab3B protein is required for long-term depression of hippocampal inhibitory synapses and for normal reversal learning - Wikidata - wikimedia - TriplyDB

Rab3B protein is required for long-term depression of hippocampal inhibitory synapses and for normal reversal learning

Rab3B protein is required for long-term depression of hippocampal inhibitory synapses and for normal reversal learning

http://www.wikidata.org/entity/Q26269894

about

The presynaptic active zone Functions of Rab Proteins at Presynaptic Sites Cell biology in neuroscience: the interplay between Hebbian and homeostatic synaptic plasticity Systems genetic analysis of hippocampal neuroanatomy and spatial learning in mice.Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.Endogenous cannabinoid signaling at inhibitory interneurons.Endocannabinoid signaling and synaptic function.Presynaptic long-term depression mediated by Gi/o-coupled receptors.Analysis of microRNA from archived formalin-fixed paraffin-embedded specimens of amyotrophic lateral sclerosis Task-specific enhancement of hippocampus-dependent learning in mice deficient in monoacylglycerol lipase, the major hydrolyzing enzyme of the endocannabinoid 2-arachidonoylglycerol A dramatic increase of C1q protein in the CNS during normal aging.Predicting the functional states of human iPSC-derived neurons with single-cell RNA-seq and electrophysiology Modulating excitation through plasticity at inhibitory synapses.Presynaptic long-term plasticity The molecular machinery of neurotransmitter release (Nobel lecture).Molecular determinants of selective dopaminergic vulnerability in Parkinson's disease: an update.An Introduction to the Endogenous Cannabinoid System.Consequences of Rab GTPase dysfunction in genetic or acquired human diseases.Astrocyte signaling controls spike timing-dependent depression at neocortical synapses.Hypertension-induced synapse loss and impairment in synaptic plasticity in the mouse hippocampus mimics the aging phenotype: implications for the pathogenesis of vascular cognitive impairment.Sex-specific Behavioral Features of Rodent Models of Autism Spectrum Disorder

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P2860

Rab3B protein is required for long-term depression of hippocampal inhibitory synapses and for normal reversal learning

http://www.wikidata.org/entity/Q26269894

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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type

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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Rab3B protein is required for ...... d for normal reversal learning

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PNAS.1112237108

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Rab3B protein is required for ...... d for normal reversal learning

@en

P2093

Chiayu Q Chiu

http://www.w3.org/2001/XMLSchema#string

Pascal S Kaeser

http://www.w3.org/2001/XMLSchema#string

Theodoros Tsetsenis

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Thomas J Younts

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P2860

The synaptic vesicle cycle

Genetic evidence for a protein-kinase-A-mediated presynaptic component in NMDA-receptor-dependent forms of long-term synaptic potentiation.

Endocannabinoid-mediated long-term plasticity requires cAMP/PKA signaling and RIM1alpha

The presynaptic active zone protein RIM1alpha is critical for normal learning and memory.

RIM determines Ca²+ channel density and vesicle docking at the presynaptic active zone

RIM proteins activate vesicle priming by reversing autoinhibitory homodimerization of Munc13

RIM proteins tether Ca2+ channels to presynaptic active zones via a direct PDZ-domain interaction

RIM1alpha phosphorylation at serine-413 by protein kinase A is not required for presynaptic long-term plasticity or learning

RIM1alpha forms a protein scaffold for regulating neurotransmitter release at the active zone

Evaluation of CB1 receptor knockout mice in the Morris water maze

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14300-5

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scholarly article

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10.1073/PNAS.1112237108

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34

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108

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Pablo E Castillo

Thomas C. Südhof

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2011-08-23T00:00:00Z

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51574051

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21844341

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3161598

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