Neuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneurons - Wikidata - wikimedia - TriplyDB

Neuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneurons

Neuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneurons

http://www.wikidata.org/entity/Q26269926

about

Autism-associated neuroligin-3 mutations commonly disrupt tonic endocannabinoid signaling Neurexins and neuroligins: recent insights from invertebrates The structure of neurexin 1α reveals features promoting a role as synaptic organizer A neural circuit for memory specificity and generalization MDGAs interact selectively with neuroligin-2 but not other neuroligins to regulate inhibitory synapse development Neuroligins/LRRTMs prevent activity- and Ca2+/calmodulin-dependent synapse elimination in cultured neurons Synaptic cell adhesion An autism-associated point mutation in the neuroligin cytoplasmic tail selectively impairs AMPA receptor-mediated synaptic transmission in hippocampus Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function Following the genes: a framework for animal modeling of psychiatric disorders Channelopathy pathogenesis in autism spectrum disorders Collagen-derived matricryptins promote inhibitory nerve terminal formation in the developing neocortex.Hippocampal neuroligin-2 overexpression leads to reduced aggression and inhibited novelty reactivity in rats Neuronal Activity Promotes Glioma Growth through Neuroligin-3 Secretion The interplay between synaptic activity and neuroligin function in the CNS A new synaptic player leading to autism risk: Met receptor tyrosine kinase The adhesion protein IgSF9b is coupled to neuroligin 2 via S-SCAM to promote inhibitory synapse development Interaction between autism-linked MDGAs and neuroligins suppresses inhibitory synapse development Genetic targeting of NRXN2 in mice unveils role in excitatory cortical synapse function and social behaviors Activity-dependent adaptations in inhibitory axons Differential regulation of the postsynaptic clustering of γ-aminobutyric acid type A (GABAA) receptors by collybistin isoforms.Developmental delays and reduced pup ultrasonic vocalizations but normal sociability in mice lacking the postsynaptic cell adhesion protein neuroligin2.Defective GABAergic neurotransmission and pharmacological rescue of neuronal hyperexcitability in the amygdala in a mouse model of fragile X syndrome Selective control of inhibitory synapse development by Slitrk3-PTPδ trans-synaptic interaction.The class 4 semaphorin Sema4D promotes the rapid assembly of GABAergic synapses in rodent hippocampus Parvalbumin-positive basket cells differentiate among hippocampal pyramidal cells.Understanding the molecular diversity of GABAergic synapses.Sex-dependent novelty response in neurexin-1α mutant mice.Differential dynamics and activity-dependent regulation of alpha- and beta-neurexins at developing GABAergic synapses Synaptic organizing complexes.Synaptic competition sculpts the development of GABAergic axo-dendritic but not perisomatic synapses Synaptic signaling and aberrant RNA splicing in autism spectrum disorders Functional fission of parvalbumin interneuron classes during fast network events GABAA receptor trafficking-mediated plasticity of inhibitory synapses.Neuroligin 2 is expressed in synapses established by cholinergic cells in the mouse brain.Fragile X syndrome: the GABAergic system and circuit dysfunction.Increased Cortical Inhibition in Autism-Linked Neuroligin-3R451C Mice Is Due in Part to Loss of Endocannabinoid Signaling Neuroligins Sculpt Cerebellar Purkinje-Cell Circuits by Differential Control of Distinct Classes of Synapses.Distinct roles of neuroligin-1 and SynCAM1 in synapse formation and function in primary hippocampal neuronal cultures Organization of central synapses by adhesion molecules.

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Neuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneurons

http://www.wikidata.org/entity/Q26269926

description

2009 nî lūn-bûn

@nan

2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

@ast

Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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type

label

Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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prefLabel

Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

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JNEUROSCI.2457-09.2009

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Journal of Neuroscience

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Neuroligin-2 deletion selectiv ...... tostatin-positive interneurons

@en

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Jay R Gibson

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Kimberly M Huber

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P2860

Neuroligins and neurexins link synaptic function to cognitive disease

Neuroligin expressed in nonneuronal cells triggers presynaptic development in contacting axons

Mutations of the X-linked genes encoding neuroligins NLGN3 and NLGN4 are associated with autism

Neurexins induce differentiation of GABA and glutamate postsynaptic specializations via neuroligins

Neurexins: synaptic cell surface proteins related to the alpha-latrotoxin receptor and laminin

X-linked mental retardation and autism are associated with a mutation in the NLGN4 gene, a member of the neuroligin family

Spontaneous subthreshold activity at motor nerve endings

CHEMOAFFINITY IN THE ORDERLY GROWTH OF NERVE FIBER PATTERNS AND CONNECTIONS

New players tip the scales in the balance between excitatory and inhibitory synapses.

Activity-dependent validation of excitatory versus inhibitory synapses by neuroligin-1 versus neuroligin-2

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scholarly article

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10.1523/JNEUROSCI.2457-09.2009

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Thomas C. Südhof

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38067700

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2814361

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