Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development? - Wikidata - wikimedia - TriplyDB

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

http://www.wikidata.org/entity/Q26738706

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Alterations in DNA methylation/demethylation intermediates predict clinical outcome in chronic lymphocytic leukemia Combining cytogenetic and epigenetic approaches in chronic lymphocytic leukemia improves prognosis prediction for patients with isolated 13q deletion.

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

http://www.wikidata.org/entity/Q26738706

description

2016 nî lūn-bûn

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2016 թուականին հրատարակուած գիտական յօդուած

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2016 թվականին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

@zh-mo

2016年論文

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2016年论文

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name

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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type

label

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

@en

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

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FONC.2016.00182

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Frontiers in Oncology

P1476

Aberrant Promoter Hypomethylation in CLL: Does It Matter for Disease Development?

@en

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Garland Michael Upchurch

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Rene Opavsky

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Staci L Haney

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P2860

DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development

A novel BTB/POZ transcriptional repressor protein interacts with the Fanconi anemia group C protein and PLZF

The protooncogene TCL1 is an Akt kinase coactivator

Exome sequencing identifies recurrent mutations of the splicing factor SF3B1 gene in chronic lymphocytic leukemia

Loss of Dnmt3b function upregulates the tumor modifier Ment and accelerates mouse lymphomagenesis

Gene silencing in cancer in association with promoter hypermethylation

Induction of tumors in mice by genomic hypomethylation

Pvt1-encoded microRNAs in oncogenesis.

Human chronic lymphocytic leukemia modeled in mouse by targeted TCL1 expression

DNMT1 is required to maintain CpG methylation and aberrant gene silencing in human cancer cells.

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182

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scholarly article

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4153034

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10.3389/FONC.2016.00182

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6

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2016-01-01T00:00:00Z

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27563627

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4980682

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