Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Advances in Neuroimmunology.Axonal chronic injury in treatment-naïve HIV+ adults with asymptomatic neurocognitive impairment and its relationship with clinical variables and cognitive status.

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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2016 nî lūn-bûn

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2016 թուականին հրատարակուած գիտական յօդուած

@hyw

2016 թվականին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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type

Item

label

Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

@en

Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

@en

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Enquan Xu

http://www.w3.org/2001/XMLSchema#string

Han Liu

http://www.w3.org/2001/XMLSchema#string

Huangui Xiong

http://www.w3.org/2001/XMLSchema#string

Jianuo Liu

http://www.w3.org/2001/XMLSchema#string

P2860

Neuregulin and BDNF induce a switch to NMDA receptor-dependent myelination by oligodendrocytes

A cofactor, TIP30, specifically enhances HIV-1 Tat-activated transcription

Human immunodeficiency virus type 1 alters brain-derived neurotrophic factor processing in neurons

Hypomyelination and increased activity of voltage-gated K(+) channels in mice lacking protein tyrosine phosphatase epsilon

HIV-associated neurocognitive disorders before and during the era of combination antiretroviral therapy: differences in rates, nature, and predictors

Blood-brain barrier and retroviral infections

Role of neurotrophic factor alterations in the neurodegenerative process in HIV associated neurocognitive disorders

LINGO-1 is a component of the Nogo-66 receptor/p75 signaling complex.

The Chronicity of HIV Infection Should Drive the Research Strategy of NeuroHIV Treatment Studies: A Critical Review

Human immunodeficiency virus type 1 and its coat protein gp120 induce apoptosis and activate JNK and ERK mitogen-activated protein kinases in human neurons

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scholarly article

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4439421

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10.3390/BRAINSCI6030023

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English

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2016-01-01T00:00:00Z

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27455335

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pathogenesis

oligodendrocyte

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5039452

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

about

P2860

P2860

Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders

description

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type

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P1433

P1476

P2093

P2860

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P3181

P356

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P577

P698

P921

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P356

P1433

P1476

P2093

P2860

P31

P3181

P356

P407

P433

P478

P577

P698

P921

P932