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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

http://www.wikidata.org/entity/Q26744695

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Analysis of Research Activity in Gastroenterology: Pancreatitis Is in Real Danger.Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2 The Interplay between Inflammation, Coagulation and Endothelial Injury in the Early Phase of Acute Pancreatitis: Clinical Implications.Restoration of energy level in the early phase of acute pediatric pancreatitis.Calcium and ATP control multiple vital functions.High versus low energy administration in the early phase of acute pancreatitis (GOULASH trial): protocol of a multicentre randomised double-blind clinical trial.Galactose protects against cell damage in mouse models of acute pancreatitis Enhanced antineoplastic/therapeutic efficacy using 5-fluorouracil-loaded calcium phosphate nanoparticles

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P2860

Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

http://www.wikidata.org/entity/Q26744695

description

2016 nî lūn-bûn

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2016 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի օգոստոսին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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type

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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prefLabel

Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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Philosophical Transactions of the Royal Society B

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Ca2+ toxicity and mitochondrial damage in acute pancreatitis: translational overview

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József Maléth

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Péter Hegyi

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Creative Commons Attribution 4.0 International

P2860

NCLX is an essential component of mitochondrial Na+/Ca2+ exchange

Orai1 is an essential pore subunit of the CRAC channel

Integrative genomics identifies MCU as an essential component of the mitochondrial calcium uniporter

A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter

STIM is a Ca2+ sensor essential for Ca2+-store-depletion-triggered Ca2+ influx

Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice.

STIM1 heteromultimerizes TRPC channels to determine their function as store-operated channels

Molecular mechanism of pancreatic and salivary gland fluid and HCO3 secretion

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

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10.1098/RSTB.2015.0425

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371

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2016-08-05T00:00:00Z

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inorganic compound

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membrane proteins

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