Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases - Wikidata - wikimedia - TriplyDB

Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

http://www.wikidata.org/entity/Q26768288

about

Stress-Induced Synaptic Dysfunction and Neurotransmitter Release in Alzheimer's Disease: Can Neurotransmitters and Neuromodulators be Potential Therapeutic Targets?The Role of Microglia in Retinal Neurodegeneration: Alzheimer's Disease, Parkinson, and Glaucoma.Antioxidant activity against H2O2-induced cytotoxicity of the ethanol extract and compounds from Pyrola decorate leaves.Glial Cells and Synaptic Plasticity.The role of TRPM2 channels in neurons, glial cells and the blood-brain barrier in cerebral ischemia and hypoxia.TRPM2 in the Brain: Role in Health and Disease

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

http://www.wikidata.org/entity/Q26768288

description

2016 nî lūn-bûn

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2016 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2016 թվականի հունվարին հրատարակված գիտական հոդված

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Neural Plasticity

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Glia and TRPM2 Channels in Plasticity of Central Nervous System and Alzheimer's Diseases

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Jing Wang

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Michael F Jackson

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Creative Commons Attribution 4.0 International

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The emerging role of glutathione in Alzheimer's disease.

Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.

Involvement of TRPM2 in peripheral nerve injury-induced infiltration of peripheral immune cells into the spinal cord in mouse neuropathic pain model.

Coexistence of two forms of LTP in ACC provides a synaptic mechanism for the interactions between anxiety and chronic pain.

TLR3 deficiency impairs spinal cord synaptic transmission, central sensitization, and pruritus in mice.

Do glia drive synaptic and cognitive impairment in disease?

Regulation of NMDA receptor subunit expression and its implications for LTD, LTP, and metaplasticity.

Glial involvement in trigeminal central sensitization.

Central sensitization: a generator of pain hypersensitivity by central neural plasticity.

Astrocyte-synapse structural plasticity.

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Alzheimer's disease

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