Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer? - Wikidata - wikimedia - TriplyDB

Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Translation from unconventional 5' start sites drives tumour initiation.Genetic divergence in the transcriptional engram of chronic alcohol abuse: A laser-capture RNA-seq study of the mouse mesocorticolimbic system.Role of Eukaryotic Initiation Factors during Cellular Stress and Cancer Progression Proteins that interact with calgranulin B in the human colon cancer cell line HCT-116.Response of Three Different Viruses to Interferon Priming and Dithiothreitol Treatment of Avian Cells.Adaptive Stress Responses During Tumor Metastasis and Dormancy.Translational Dysregulation in Cancer: Molecular Insights and Potential Clinical Applications in Biomarker Development.Two distinct nodes of translational inhibition in the Integrated Stress Response.A Unique ISR Program Determines Cellular Responses to Chronic Stress.Crystal Structure of the C-terminal Domain of Human eIF2D and Its Implications on Eukaryotic Translation Initiation.Translation acrobatics: how cancer cells exploit alternate modes of translational initiation

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

http://www.wikidata.org/entity/Q26774488

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2015 nî lūn-bûn

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2015 թուականին հրատարակուած գիտական յօդուած

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2015 թվականին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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type

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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FONC.2015.00264

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Frontiers in Oncology

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Could the eIF2α-Independent Translation Be the Achilles Heel of Cancer?

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Martin Holcik

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A novel candidate oncogene, MCT-1, is involved in cell cycle progression

Activities of Ligatin and MCT-1/DENR in eukaryotic translation initiation and ribosomal recycling

Regulation of XIAP translation and induction by MDM2 following irradiation

GTP-independent tRNA delivery to the ribosomal P-site by a novel eukaryotic translation factor

Targeting MCT-1 oncogene inhibits Shc pathway and xenograft tumorigenicity

XIAP deficiency: a unique primary immunodeficiency best classified as X-linked familial hemophagocytic lymphohistiocytosis and not as X-linked lymphoproliferative disease

Autophosphorylation sites participate in the activation of the double-stranded-RNA-activated protein kinase PKR

eIF2-dependent and eIF2-independent modes of initiation on the CSFV IRES: a common role of domain II

Structure of the mammalian 80S initiation complex with initiation factor 5B on HCV-IRES RNA

Internal initiation of translation of eukaryotic mRNA directed by a sequence derived from poliovirus RNA

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