Controlling DNA replication origins in response to DNA damage - inhibit globally, activate locally
about
Activation of new replication foci under conditions of replication stress.Regulation and Function of Cdt1; A Key Factor in Cell Proliferation and Genome StabilityA concomitant loss of dormant origins and FANCC exacerbates genome instability by impairing DNA replication fork progression.Recombinase and translesion DNA polymerase decrease the speed of replication fork progression during the DNA damage response in Escherichia coli cells.Tight Chk1 Levels Control Replication Cluster Activation in Xenopus.Allele-specific analysis of DNA replication origins in mammalian cells.Catalytic strand separation by RECQ1 is required for RPA-mediated response to replication stress.Chromatin-associated degradation is defined by UBXN-3/FAF1 to safeguard DNA replication fork progression.Interaction of RECQ4 and MCM10 is important for efficient DNA replication origin firing in human cells.The FACT histone chaperone guides histone H4 into its nucleosomal conformation in Saccharomyces cerevisiae.The DNA translocase FANCM/MHF promotes replication traverse of DNA interstrand crosslinks.The Intra-S Checkpoint Responses to DNA Damage.The essential kinase ATR: ensuring faithful duplication of a challenging genome.The Replication of Frataxin Gene Is Assured by Activation of Dormant Origins in the Presence of a GAA-Repeat Expansion.Strategic role of the ubiquitin-dependent segregase p97 (VCP or Cdc48) in DNA replication.PrimPol-A new polymerase on the block.Interaction of Chk1 with Treslin negatively regulates the initiation of chromosomal DNA replication.Replication stalling and DNA microsatellite instability.A Critical Balance: dNTPs and the Maintenance of Genome StabilityThe Role of the Transcriptional Response to DNA Replication Stress.DNA Replication Origins and Fork Progression at Mammalian TelomeresDormant origins as a built-in safeguard in eukaryotic DNA replication against genome instability and disease development.Targeting DNA repair and replication stress in the treatment of ovarian cancer.Overexpression of the Replicative Helicase in Escherichia coli Inhibits Replication Initiation and Replication Fork Reloading.Mcm2-7 Is an Active Player in the DNA Replication Checkpoint Signaling Cascade via Proposed Modulation of Its DNA Gate.Mitomycin C reduces abundance of replication forks but not rates of fork progression in primary and transformed human cells.Set2 Methyltransferase Facilitates DNA Replication and Promotes Genotoxic Stress Responses through MBF-Dependent Transcription.Origin licensing requires ATP binding and hydrolysis by the MCM replicative helicase.A conserved Polϵ binding module in Ctf18-RFC is required for S-phase checkpoint activation downstream of Mec1.Inter-Fork Strand Annealing causes genomic deletions during the termination of DNA replication.The CDK-PLK1 axis targets the DNA damage checkpoint sensor protein RAD9 to promote cell proliferation and tolerance to genotoxic stress.Orchestration of DNA Damage Checkpoint Dynamics across the Human Cell Cycle.Dormant origins and fork protection mechanisms rescue sister forks arrested by transcription.The need to regulate replication fork speed.Transcription-Replication Conflict Orientation Modulates R-Loop Levels and Activates Distinct DNA Damage ResponsesRecent advances in understanding DNA replication: cell type-specific adaptation of the DNA replication program
P2860
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P2860
Controlling DNA replication origins in response to DNA damage - inhibit globally, activate locally
description
2013 nî lūn-bûn
@nan
2013 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մարտին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
@wuu
name
Controlling DNA replication or ...... bit globally, activate locally
@ast
Controlling DNA replication or ...... bit globally, activate locally
@en
Controlling DNA replication or ...... bit globally, activate locally
@nl
type
label
Controlling DNA replication or ...... bit globally, activate locally
@ast
Controlling DNA replication or ...... bit globally, activate locally
@en
Controlling DNA replication or ...... bit globally, activate locally
@nl
prefLabel
Controlling DNA replication or ...... bit globally, activate locally
@ast
Controlling DNA replication or ...... bit globally, activate locally
@en
Controlling DNA replication or ...... bit globally, activate locally
@nl
P3181
P356
P1476
Controlling DNA replication or ...... bit globally, activate locally
@en
P2093
Mona Yekezare
P304
P3181
P356
10.1242/JCS.096701
P407
P577
2013-03-01T00:00:00Z