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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

http://www.wikidata.org/entity/Q27003067

about

Platelets in Pulmonary Immune Responses and Inflammatory Lung Diseases Electrical muscle stimulation in thomboprophylaxis: review and a derived hypothesis about thrombogenesis-the 4th factor Markers of coagulation and inflammation often remain elevated in ART-treated HIV-infected patients Salivary Thromboxane A2-Binding Proteins from Triatomine Vectors of Chagas Disease Inhibit Platelet-Mediated Neutrophil Extracellular Traps (NETs) Formation and Arterial Thrombosis From basic mechanisms to clinical applications in heart protection, new players in cardiovascular diseases and cardiac theranostics: meeting report from the third international symposium on "New frontiers in cardiovascular research"Plasmodium falciparum infection induces expression of a mosquito salivary protein (Agaphelin) that targets neutrophil function and inhibits thrombosis without impairing hemostasis Coagulation and morbidity in treated HIV infection.Neutrophil extracellular traps form predominantly during the organizing stage of human venous thromboembolism development.Endovascular laser–tissue interactions and biological responses in relation to endovenous laser therapy.Histopathological evaluation of thrombus in patients presenting with stent thrombosis. A multicenter European study: a report of the prevention of late stent thrombosis by an interdisciplinary global European effort consortium The simultaneous occurrence of both hypercoagulability and hypofibrinolysis in blood and serum during systemic inflammation, and the roles of iron and fibrin(ogen).Fibronectin maintains the balance between hemostasis and thrombosis.Propagation of thrombosis by neutrophils and extracellular nucleosome networks.Venous thromboembolism: thrombosis, inflammation, and immunothrombosis for clinicians.Tumor-Derived Exosomes Induce the Formation of Neutrophil Extracellular Traps: Implications For The Establishment of Cancer-Associated Thrombosis.Disulfide HMGB1 derived from platelets coordinates venous thrombosis in mice.The saponin D39 blocks dissociation of non-muscular myosin heavy chain IIA from TNF receptor 2, suppressing tissue factor expression and venous thrombosis.Stenosis of the Inferior Vena Cava: A Murine Model of Deep Vein Thrombosis.Tissue factor-bearing microparticles and inflammation: a potential mechanism for the development of venous thromboembolism in cancer.Natural antibodies to oxidation-specific epitopes: innate immune response and venous thromboembolic disease.NMMHC IIA inhibition impedes tissue factor expression and venous thrombosis via Akt/GSK3β-NF-κB signalling pathways in the endothelium.

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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2013 nî lūn-bûn

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2013 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2013 թվականի հունիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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Journal of Thrombosis and Haemostasis

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Crossroads of coagulation and innate immunity: the case of deep vein thrombosis

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P2093

B. Engelmann

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C. Schulz

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S. Massberg

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P2860

Neutrophil extracellular traps promote deep vein thrombosis in mice

Leukocytes and the natural history of deep vein thrombosis: current concepts and future directions

Extracellular DNA traps promote thrombosis.

GMP-140, a platelet alpha-granule membrane protein, is also synthesized by vascular endothelial cells and is localized in Weibel-Palade bodies

Platelet polyphosphates are proinflammatory and procoagulant mediators in vivo

Neutrophil extracellular traps kill bacteria

Hereditary neutropenia: dogs explain human neutrophil elastase mutations

Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood

Neutrophil extracellular traps capture and kill Candida albicans yeast and hyphal forms

Getting to the site of inflammation: the leukocyte adhesion cascade updated

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233-41

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scholarly article

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505999

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10.1111/JTH.12261

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11 Suppl 1

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2013-06-01T00:00:00Z

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264370079

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23809127

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