Rho GTPases and their roles in cancer metabolism - Wikidata - wikimedia - TriplyDB

Rho GTPases and their roles in cancer metabolism

Rho GTPases and their roles in cancer metabolism

http://www.wikidata.org/entity/Q27003967

about

CDC42 Use in Viral Cell Entry Processes by RNA Viruses Deleted in liver cancer-1 (DLC1): an emerging metastasis suppressor gene DLC1 suppresses NF-κB activity in prostate cancer cells due to its stabilizing effect on adherens junctions TNF-α promotes extracellular vesicle release in mouse astrocytes through glutaminase Membrane phospholipid redistribution in cancer micro-particles and implications in the recruitment of cationic protein factors.δ-Catenin, a Wnt/β-catenin modulator, reveals inducible mutagenesis promoting cancer cell survival adaptation and metabolic reprogramming.Cancerous epithelial cell lines shed extracellular vesicles with a bimodal size distribution that is sensitive to glutamine inhibition Cdc42p-interacting protein Bem4p regulates the filamentous-growth mitogen-activated protein kinase pathway.Simultaneously targeting tissue transglutaminase and kidney type glutaminase sensitizes cancer cells to acid toxicity and offers new opportunities for therapeutic intervention ERG oncoprotein inhibits ANXA2 expression and function in prostate cancer Association between GWAS-identified genetic variations and disease prognosis for patients with colorectal cancer.Forkhead Box F1 promotes breast cancer cell migration by upregulating lysyl oxidase and suppressing Smad2/3 signaling Breast cancer-derived extracellular vesicles stimulate myofibroblast differentiation and pro-angiogenic behavior of adipose stem cells.Spatial landmarks regulate a Cdc42-dependent MAPK pathway to control differentiation and the response to positional compromise.Stress granules and cell signaling: more than just a passing phase?Therapeutic strategies impacting cancer cell glutamine metabolism.The sarcomeric M-region: a molecular command center for diverse cellular processes.Targeting Glutamine Induces Apoptosis: A Cancer Therapy Approach.Effects of Ras homolog gene family, member C gene silencing combined with rapamycin on hepatocellular carcinoma cell growth.Biology and biogenesis of shed microvesicles.Rac1 GTPase-deficient HeLa cells present reduced DNA repair, proliferation, and survival under UV or gamma irradiation.Rac1 and Cdc42 Play Important Roles in Arsenic Neurotoxicity in Primary Cultured Rat Cerebellar Astrocytes.Shedding light on the cell biology of extracellular vesicles.Glutamine metabolism via glutaminase 1 in autosomal-dominant polycystic kidney disease.Identification of a Fragmented Small GTPase Capable of Conditional Effector Binding.Design of novel lead molecules against RhoG protein as cancer target - a computational study.Glutamate enrichment as new diagnostic opportunity in breast cancer.Myc requires RhoA/SRF to reprogram glutamine metabolism.

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P2860

Rho GTPases and their roles in cancer metabolism

http://www.wikidata.org/entity/Q27003967

description

2013 nî lūn-bûn

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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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Rho GTPases and their roles in cancer metabolism

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J.MOLMED.2012.10.011

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Trends in Molecular Medicine

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Rho GTPases and their roles in cancer metabolism

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P2093

Jon W. Erickson

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Kristin F. Wilson

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Marc A. Antonyak

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Richard A. Cerione

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P2860

DLC-1 gene inhibits human breast cancer cell growth and in vivo tumorigenicity

Full-length human glutaminase in complex with an allosteric inhibitor

Exome sequencing identifies recurrent somatic RAC1 mutations in melanoma

Glioblastoma microvesicles transport RNA and proteins that promote tumour growth and provide diagnostic biomarkers

Microvesicles: mediators of extracellular communication during cancer progression

A pleiotropically acting microRNA, miR-31, inhibits breast cancer metastasis

Endogenous, hyperactive Rac3 controls proliferation of breast cancer cells by a p21-activated kinase-dependent pathway

Characterization of RhoC expression in benign and malignant breast disease: a potential new marker for small breast carcinomas with metastatic ability

Pyruvate kinase M2 is a phosphotyrosine-binding protein

Structural basis for the allosteric inhibitory mechanism of human kidney-type glutaminase (KGA) and its regulation by Raf-Mek-Erk signaling in cancer cell metabolism

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74-82

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scholarly article

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10.1016/J.MOLMED.2012.10.011

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3607349

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