Gene therapy targets in heart failure: the path to translation - Wikidata - wikimedia - TriplyDB

Gene therapy targets in heart failure: the path to translation

Gene therapy targets in heart failure: the path to translation

http://www.wikidata.org/entity/Q27022444

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Heart Failure: Diagnosis, Management and Utilization Prospects for gene transfer for clinical heart failure Structures of the Excited States of Phospholamban and Shifts in Their Populations upon Phosphorylation Targeting S100A1 in heart failure Engineering Parvalbumin for the Heart: Optimizing the Mg Binding Properties of Rat β-Parvalbumin Gene transfer for congestive heart failure: update 2013.

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Gene therapy targets in heart failure: the path to translation

http://www.wikidata.org/entity/Q27022444

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Gene therapy targets in heart failure: the path to translation

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Clinical Pharmacology & Therapeutics

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Gene therapy targets in heart failure: the path to translation

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H A Katus

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H Tscheschner

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J Reinkober

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J Ritterhoff

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P Most

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P W J Raake

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W J Koch

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P2860

Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a.

A mutation in the human phospholamban gene, deleting arginine 14, results in lethal, hereditary cardiomyopathy.

Targeted beta-adrenergic receptor kinase (betaARK1) inhibition by gene transfer in failing human hearts

S100A1: a regulator of myocardial contractility

Myocardial adeno-associated virus serotype 6-betaARKct gene therapy improves cardiac function and normalizes the neurohormonal axis in chronic heart failure

Preservation of myocardial beta-adrenergic receptor signaling delays the development of heart failure after myocardial infarction

Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathy

Transgenic overexpression of the Ca2+-binding protein S100A1 in the heart leads to increased in vivo myocardial contractile performance

Cardiac excitation-contraction coupling

Inhibition of protein phosphatase 1 by inhibitor-2 gene delivery ameliorates heart failure progression in genetic cardiomyopathy

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10.1038/CLPT.2011.148

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