Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
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Marijuana: current concepts(†)Cannabinoid-based drugs targeting CB1 and TRPV1, the sympathetic nervous system, and arthritisVascular targets for cannabinoids: animal and human studiesMedical consequences of marijuana use: a review of current literatureEffects of the endogenous cannabinoid anandamide on voltage-dependent sodium and calcium channels in rat ventricular myocytesPharmacological profiling of the hemodynamic effects of cannabinoid ligands: a combined in vitro and in vivo approachSuccessful TRPV1 antagonist treatment for cardiac hypertrophy and heart failure in miceMitogen-Activated Protein Kinase and Intracellular Polyamine Signaling Is Involved in TRPV1 Activation-Induced Cardiac HypertrophyMechanisms of endothelium-dependent relaxation evoked by anandamide in isolated human pulmonary arteries.Endocannabinoid system as a potential mechanism for n-3 long-chain polyunsaturated fatty acid mediated cardiovascular protection.Cannabinoids and the Kidney: Effects in Health and Disease.CB1 receptor activation in the rat paraventricular nucleus induces bi-directional cardiovascular effects via modification of glutamatergic and GABAergic neurotransmission.Role of cannabis in cardiovascular disorders.Involvement of endocannabinoid neurotransmission in the bed nucleus of stria terminalis in cardiovascular responses to acute restraint stress in rats.A randomised controlled cross-over double-blind pilot study protocol on THC:CBD oromucosal spray efficacy as an add-on therapy for post-stroke spasticity.Blood pressure-lowering effects of propofol or sevoflurane anaesthesia are not due to enhanced nitric oxide formation or bioavailability.Endocannabinoids modulate Gq/11 protein-coupled receptor agonist-induced vasoconstriction via a negative feedback mechanism.Do cannabinoids exhibit a tyramine-like effect?Chronic inhibition of fatty acid amide hydrolase by URB597 produces differential effects on cardiac performance in normotensive and hypertensive rats.Blockade of cannabinoid receptors reduces inflammation, leukocyte accumulation and neovascularization in a model of sponge-induced inflammatory angiogenesis.Activation of CB1 receptors by 2-arachidonoylglycerol attenuates vasoconstriction induced by U46619 and angiotensin II in human and rat pulmonary arteries.Synthetic cannabinoids found in "spice" products alter body temperature and cardiovascular parameters in conscious male rats.A Systematic Review of the Complex Effects of Cannabinoids on Cerebral and Peripheral Circulation in Animal Models.
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P2860
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
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2012 nî lūn-bûn
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2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2012 թվականի ապրիլին հրատարակված գիտական հոդված
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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name
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@ast
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@en
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@nl
type
label
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@ast
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@en
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@nl
prefLabel
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@ast
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@en
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@nl
P2860
P3181
P1476
Triphasic blood pressure responses to cannabinoids: do we understand the mechanism?
@en
P2093
Eberhard Schlicker
P2860
P304
P3181
P356
10.1111/J.1476-5381.2011.01747.X
P407
P577
2012-04-01T00:00:00Z