Beta-catenin signaling plays a disparate role in different phases of fracture repair: implications for therapy to improve bone healing - Wikidata - wikimedia - TriplyDB

Beta-catenin signaling plays a disparate role in different phases of fracture repair: implications for therapy to improve bone healing

Beta-catenin signaling plays a disparate role in different phases of fracture repair: implications for therapy to improve bone healing

http://www.wikidata.org/entity/Q27307639

about

Developmental Drift and the Role of Wnt Signaling in Aging Targeting the Wnt pathways for therapies Cutaneous wound healing: recruiting developmental pathways for regeneration Current insights on the regenerative potential of the periosteum: molecular, cellular, and endogenous engineering approaches Recent advances in nano scaffolds for bone repair Biological and molecular profile of fracture non-union tissue: current insights The Wnt serpentine receptor Frizzled-9 regulates new bone formation in fracture healing Gene expression dynamics during bone healing and osseointegration Dickkopf-1 regulates bone formation in young growing rodents and upon traumatic injury Influence of single and binary doping of strontium and lithium on in vivo biological properties of bioactive glass scaffolds Beta-catenin and transforming growth factor beta have distinct roles regulating fibroblast cell motility and the induction of collagen lattice contraction.Modulation of Wnt signaling influences fracture repair Wnt/β-catenin pathway regulates cementogenic differentiation of adipose tissue-deprived stem cells in dental follicle cell-conditioned medium Increased skeletal VEGF enhances beta-catenin activity and results in excessively ossified bones.Increased Runx2 expression associated with enhanced Wnt signaling in PDLLA internal fixation for fracture treatment.An appropriate Wnt/β-catenin expression level during the remodeling phase is required for improved bone fracture healing in mice Exogenous activation of Wnt/β-catenin signaling attenuates binge alcohol-induced deficient bone fracture healing.Upregulation of inflammatory genes and downregulation of sclerostin gene expression are key elements in the early phase of fragility fracture healing.The spatiotemporal role of COX-2 in osteogenic and chondrogenic differentiation of periosteum-derived mesenchymal progenitors in fracture repair.Aging periosteal progenitor cells have reduced regenerative responsiveness to bone injury and to the anabolic actions of PTH 1-34 treatment.Osteoblast-specific Krm2 overexpression and Lrp5 deficiency have different effects on fracture healing in mice.Current concepts in the pathogenesis of traumatic temporomandibular joint ankylosis Emerging therapeutic opportunities for skeletal restoration.A high throughput screen identifies Nefopam as targeting cell proliferation in β-catenin driven neoplastic and reactive fibroproliferative disorders.T-lymphocytes enable osteoblast maturation via IL-17F during the early phase of fracture repair Activation of the Hh pathway in periosteum-derived mesenchymal stem cells induces bone formation in vivo: implication for postnatal bone repair.Cellular mechanisms of bone remodeling Parameters for lithium treatment are critical in its enhancement of fracture-healing in rodents.Effects of constitutive β-catenin activation on vertebral bone growth and remodeling at different postnatal stages in mice Inhibition of beta-catenin signaling by Pb leads to incomplete fracture healing Inhibition of GSK-3β rescues the impairments in bone formation and mechanical properties associated with fracture healing in osteoblast selective connexin 43 deficient mice.Alcohol exposure and mechanisms of tissue injury and repair.Role of WNT16 in the regulation of periosteal bone formation in female mice.β-catenin enhances odontoblastic differentiation of dental pulp cells through activation of Runx2.A rare human syndrome provides genetic evidence that WNT signaling is required for reprogramming of fibroblasts to induced pluripotent stem cells Transient gamma-secretase inhibition accelerates and enhances fracture repair likely via Notch signaling modulation Antioxidant therapy attenuates deficient bone fracture repair associated with binge alcohol exposure.Role of Wnt signaling in fracture healing Anti-DKK1 antibody promotes bone fracture healing through activation of β-catenin signaling.Dkk1 haploinsufficiency requires expression of Bmp2 for bone anabolic activity

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Beta-catenin signaling plays a disparate role in different phases of fracture repair: implications for therapy to improve bone healing

http://www.wikidata.org/entity/Q27307639

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2007年の論文

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Axin, a negative regulator of the Wnt signaling pathway, forms a complex with GSK-3beta and beta-catenin and promotes GSK-3beta-dependent phosphorylation of beta-catenin.

Intestinal polyposis in mice with a dominant stable mutation of the beta-catenin gene

WISP-1 is an osteoblastic regulator expressed during skeletal development and fracture repair

Regulation of LEF-1/TCF transcription factors by Wnt and other signals

LDL receptor-related protein 5 (LRP5) affects bone accrual and eye development

High bone density due to a mutation in LDL-receptor-related protein 5

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Canonical Wnt signaling in differentiated osteoblasts controls osteoclast differentiation

Canonical Wnt/beta-catenin signaling prevents osteoblasts from differentiating into chondrocytes

Sequential roles of Hedgehog and Wnt signaling in osteoblast development

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