Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse
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Resveratrol and MyopathySIRT1: A Novel Target for the Treatment of Muscular DystrophiesMetabogenic and Nutriceutical Approaches to Address Energy Dysregulation and Skeletal Muscle Wasting in Duchenne Muscular DystrophyPre-clinical drug tests in the mdx mouse as a model of dystrophinopathies: an overviewNew insights into PGC-1 coactivators: redefining their role in the regulation of mitochondrial function and beyondMuscle-specific SIRT1 gain-of-function increases slow-twitch fibers and ameliorates pathophysiology in a mouse model of duchenne muscular dystrophyEffects of resveratrol on the recovery of muscle mass following disuse in the plantaris muscle of aged ratsNutraceuticals and Their Potential to Treat Duchenne Muscular Dystrophy: Separating the Credible from the ConjectureAnimal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapyMetformin protects skeletal muscle from cardiotoxin induced degenerationLong-Term Quercetin Dietary Enrichment Partially Protects Dystrophic Skeletal MuscleTWEAK/Fn14, a pathway and novel therapeutic target in myotonic dystrophy.Muscle structure influences utrophin expression in mdx mice.Resveratrol induces expression of the slow, oxidative phenotype in mdx mouse muscle together with enhanced activity of the SIRT1-PGC-1α axis.Oxidative damage in the gastrocnemius of patients with peripheral artery disease is myofiber type selective.Systemic down-regulation of delta-9 desaturase promotes muscle oxidative metabolism and accelerates muscle function recovery following nerve injury.Porcine models of muscular dystrophyMetabolic remodeling agents show beneficial effects in the dystrophin-deficient mdx mouse modelEffect of genetic background on the dystrophic phenotype in mdx mice.Perm1 enhances mitochondrial biogenesis, oxidative capacity, and fatigue resistance in adult skeletal muscle.Defects in mitochondrial localization and ATP synthesis in the mdx mouse model of Duchenne muscular dystrophy are not alleviated by PDE5 inhibitionLoss of cIAP1 attenuates soleus muscle pathology and improves diaphragm function in mdx mice.Muscle-Specific Myosin Heavy Chain Shifts in Response to a Long-Term High Fat/High Sugar Diet and Resveratrol Treatment in Nonhuman Primates.Abnormally Small Neuromuscular Junctions in the Extraocular Muscles From Subjects With Idiopathic Nystagmus and Nystagmus Associated With Albinism.Trip12, a HECT domain E3 ubiquitin ligase, targets Sox6 for proteasomal degradation and affects fiber type-specific gene expression in muscle cells.Prospect of gene therapy for cardiomyopathy in hereditary muscular dystrophy.Distribution of myosin heavy chain isoforms in muscular dystrophy: insights into disease pathology.Long-term wheel running compromises diaphragm function but improves cardiac and plantarflexor function in the mdx mouseComparative Genomics of X-linked Muscular Dystrophies: The Golden Retriever ModelBlunted angiogenesis and hypertrophy are associated with increased fatigue resistance and unchanged aerobic capacity in old overloaded mouse muscle.Coupling of mitochondrial function and skeletal muscle fiber type by a miR-499/Fnip1/AMPK circuit.PGC-1α modulates necrosis, inflammatory response, and fibrotic tissue formation in injured skeletal muscle.Molecular mechanism of sphingosine-1-phosphate action in Duchenne muscular dystrophy.Mitochondria mediate cell membrane repair and contribute to Duchenne muscular dystrophy.Recessive and dominant mutations in COL12A1 cause a novel EDS/myopathy overlap syndrome in humans and mice.Turn up the power - pharmacological activation of mitochondrial biogenesis in mouse models.Increased AMP-activated protein kinase in skeletal muscles of Murphy Roth Large mice and its potential role in altered metabolism.Role of PGC-1α signaling in skeletal muscle health and disease.Duchenne muscular dystrophy drug discovery - the application of utrophin promoter activation screening.Mechanisms for fiber-type specificity of skeletal muscle atrophy.
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P2860
Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse
description
2012 nî lūn-bûn
@nan
2012 թուականին հրատարակուած գիտական յօդուած
@hyw
2012 թվականին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@ast
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@en
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@nl
type
label
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@ast
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@en
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@nl
prefLabel
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@ast
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@en
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@nl
P2093
P2860
P3181
P1433
P1476
Rescue of dystrophic skeletal ...... er type shift in the mdx mouse
@en
P2093
Elisabeth R Barton
H Lee Sweeney
Joshua T Selsby
Kevin J Morine
Klara Pendrak
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0030063
P407
P577
2012-01-01T00:00:00Z