GDNF is not required for catecholaminergic neuron survival in vivo - Wikidata - wikimedia - TriplyDB

GDNF is not required for catecholaminergic neuron survival in vivo

GDNF is not required for catecholaminergic neuron survival in vivo

http://www.wikidata.org/entity/Q27312615

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GDNF Overexpression from the Native Locus Reveals its Role in the Nigrostriatal Dopaminergic System Function GDNF-Ret signaling in midbrain dopaminergic neurons and its implication for Parkinson disease Striatal GDNF Production Is Independent to Circulating Estradiol Level Despite Pan-Neuronal Activation in the Female Mouse Compromised Survival of Cerebellar Molecular Layer Interneurons Lacking GDNF Receptors GFRα1 or RET Impairs Normal Cerebellar Motor Learning.A regulatable AAV vector mediating GDNF biological effects at clinically-approved sub-antimicrobial doxycycline doses.Glial Cell Line-Derived Neurotrophic Factor Gene Delivery in Parkinson's Disease: A Delicate Balance between Neuroprotection, Trophic Effects, and Unwanted Compensatory Mechanisms.Therapeutic potential of the endoplasmic reticulum located and secreted CDNF/MANF family of neurotrophic factors in Parkinson's disease.Current disease modifying approaches to treat Parkinson's disease.Dopaminergic Neurons and Brain Reward Pathways: From Neurogenesis to Circuit Assembly.Ret is essential to mediate GDNF's neuroprotective and neuroregenerative effect in a Parkinson disease mouse model.Update of neurotrophic factors in neurobiology of addiction and future directions.The neuroprotective and regenerative potential of parkin and GDNF/Ret signaling in the midbrain dopaminergic system.Exercise-Induced Neuroprotection of the Nigrostriatal Dopamine System in Parkinson's Disease.TNF superfamily member APRIL enhances midbrain dopaminergic axon growth and contributes to the nigrostriatal projection in vivo.Obesity: Receptors identified for a weight regulator.Pre-α-pro-GDNF and Pre-β-pro-GDNF Isoforms Are Neuroprotective in the 6-hydroxydopamine Rat Model of Parkinson's Disease.

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GDNF is not required for catecholaminergic neuron survival in vivo

http://www.wikidata.org/entity/Q27312615

description

2015 nî lūn-bûn

@nan

2015 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

GDNF is not required for catecholaminergic neuron survival in vivo

@ast

GDNF is not required for catecholaminergic neuron survival in vivo

@en

GDNF is not required for catecholaminergic neuron survival in vivo

@nl

type

label

GDNF is not required for catecholaminergic neuron survival in vivo

@ast

GDNF is not required for catecholaminergic neuron survival in vivo

@en

GDNF is not required for catecholaminergic neuron survival in vivo

@nl

prefLabel

GDNF is not required for catecholaminergic neuron survival in vivo

@ast

GDNF is not required for catecholaminergic neuron survival in vivo

@en

GDNF is not required for catecholaminergic neuron survival in vivo

@nl

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Nature Neuroscience

P1476

GDNF is not required for catecholaminergic neuron survival in vivo

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P2093

Carolina Vilenius

http://www.w3.org/2001/XMLSchema#string

Jesse Lindholm

http://www.w3.org/2001/XMLSchema#string

Kärt Varendi

http://www.w3.org/2001/XMLSchema#string

Mari-Anne Härma

http://www.w3.org/2001/XMLSchema#string

Mart Saarma

http://www.w3.org/2001/XMLSchema#string

P2860

Absence of Ret signaling in mice causes progressive and late degeneration of the nigrostriatal system

GDNF: a glial cell line-derived neurotrophic factor for midbrain dopaminergic neurons

The GDNF family: signalling, biological functions and therapeutic value

Disruption of the glucocorticoid receptor gene in the nervous system results in reduced anxiety

Characterisation of behavioural and neurodegenerative changes induced by intranigral 6-hydroxydopamine lesions in a mouse model of Parkinson's disease.

Tamoxifen-independent recombination in the RIP-CreER mouse.

Dysregulated dopamine storage increases the vulnerability to α-synuclein in nigral neurons.

Priorities in Parkinson's disease research.

The new stereological tools: disector, fractionator, nucleator and point sampled intercepts and their use in pathological research and diagnosis.

Unilateral nigrostriatal 6-hydroxydopamine lesions in mice I: motor impairments identify extent of dopamine depletion at three different lesion sites.

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319-322

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scholarly article

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2977072

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10.1038/NN.3941

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3

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18

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Petteri Piepponen

Anders Björklund

Jaan-Olle Andressoo

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2015-03-01T00:00:00Z

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272823087

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1031225401

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25710828

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4878654

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