Apoptosis is essential for neutrophil functional shutdown and determines tissue damage in experimental pneumococcal meningitis - Wikidata - wikimedia - TriplyDB

Apoptosis is essential for neutrophil functional shutdown and determines tissue damage in experimental pneumococcal meningitis

Apoptosis is essential for neutrophil functional shutdown and determines tissue damage in experimental pneumococcal meningitis

http://www.wikidata.org/entity/Q27316800

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Proliferating cell nuclear antigen acts as a cytoplasmic platform controlling human neutrophil survival Defense at the border: the blood-brain barrier versus bacterial foreigners Inflammasome/IL-1β Responses to Streptococcal Pathogens Apoptotic cell clearance: basic biology and therapeutic potential.Host-pathogen interactions in bacterial meningitis Induction of eosinophil apoptosis by the cyclin-dependent kinase inhibitor AT7519 promotes the resolution of eosinophil-dominant allergic inflammation NLRP3 Inflammasome in Neurological Diseases, from Functions to Therapies Multi-scale optical imaging of the delayed type hypersensitivity reaction attenuated by rapamycin Leukocyte attraction by CCL20 and its receptor CCR6 in humans and mice with pneumococcal meningitis.Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.The p53-target gene puma drives neutrophil-mediated protection against lethal bacterial sepsis Harnessing Neutrophil Survival Mechanisms during Chronic Infection by Pseudomonas aeruginosa: Novel Therapeutic Targets to Dampen Inflammation in Cystic Fibrosis Kinase activity profiling of pneumococcal pneumonia Type I interferon production induced by Streptococcus pyogenes-derived nucleic acids is required for host protection Reduced neutrophil apoptosis in diabetic mice during staphylococcal infection leads to prolonged Tnfα production and reduced neutrophil clearance.Programmed cell death and its role in inflammation siRNA-mediated gene silencing of MexB from the MexA-MexB-OprM efflux pump in Pseudomonas aeruginosa Neutrophil apoptosis: relevance to the innate immune response and inflammatory disease.Neutrophil granulocytes recruited upon translocation of intestinal bacteria enhance graft-versus-host disease via tissue damage.Inhibition of matrix metalloproteinases attenuates brain damage in experimental meningococcal meningitis.Roscovitine-induced apoptosis in neutrophils and neutrophil progenitors is regulated by the Bcl-2-family members Bim, Puma, Noxa and Mcl-1 IL-32γ delays spontaneous apoptosis of human neutrophils through MCL-1, regulated primarily by the p38 MAPK pathway.Complement component 5 contributes to poor disease outcome in humans and mice with pneumococcal meningitis.Francisella tularensis inhibits the intrinsic and extrinsic pathways to delay constitutive apoptosis and prolong human neutrophil lifespan.Period of irreversible therapeutic intervention during sepsis correlates with phase of innate immune dysfunction.Resolvin E1 promotes phagocytosis-induced neutrophil apoptosis and accelerates resolution of pulmonary inflammation.R-roscovitine reduces lung inflammation induced by lipoteichoic acid and Streptococcus pneumoniae.Flavones induce neutrophil apoptosis by down-regulation of Mcl-1 via a proteasomal-dependent pathway Modulating Innate and Adaptive Immunity by (R)-Roscovitine: Potential Therapeutic Opportunity in Cystic Fibrosis.Francisella tularensis Modulates a Distinct Subset of Regulatory Factors and Sustains Mitochondrial Integrity to Impair Human Neutrophil Apoptosis.Roscovitine ameliorates endotoxin-induced uveitis through neutrophil apoptosis.Genetic and pharmacological inhibition of CDK9 drives neutrophil apoptosis to resolve inflammation in zebrafish in vivo.Neutrophils: potential therapeutic targets in tularemia?Toll-like receptor 9 signaling delays neutrophil apoptosis by increasing transcription of Mcl-1.Cyclin-dependent kinase inhibitor drugs as potential novel anti-inflammatory and pro-resolution agents.Downregulation of Mcl-1 has anti-inflammatory pro-resolution effects and enhances bacterial clearance from the lung Cyclin-dependent kinase inhibitors: a survey of recent patent literature.Bacterial meningitis: current therapy and possible future treatment options.Treatment of bacterial meningitis: an update.Modulation of Neutrophil Apoptosis and the Resolution of Inflammation through β2 Integrins.

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Apoptosis is essential for neutrophil functional shutdown and determines tissue damage in experimental pneumococcal meningitis

http://www.wikidata.org/entity/Q27316800

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2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Apoptosis is essential for neu ...... mental pneumococcal meningitis

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Bianca Obermaier

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Georg Häcker

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Hans Häcker

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Robert Paul

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Susanne Kirschnek

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Tobias Frankenberg

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Uwe Koedel

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P2860

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

Cyclin-dependent kinase inhibitors enhance the resolution of inflammation by promoting inflammatory cell apoptosis

Resolution of inflammation: the beginning programs the end

Doxycycline reduces mortality and injury to the brain and cochlea in experimental pneumococcal meningitis.

Shutdown of an acute T cell immune response to viral infection is mediated by the proapoptotic Bcl-2 homology 3-only protein Bim.

Bacterial meningitis--a view of the past 90 years.

Molecular control of neutrophil apoptosis.

Bim mediates apoptosis of CD127(lo) effector T cells and limits T cell memory.

Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages

Pathogenesis and pathophysiology of pneumococcal meningitis.

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26249994

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19478887

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apoptotic process

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2682662

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