HIF1A reduces acute lung injury by optimizing carbohydrate metabolism in the alveolar epithelium - Wikidata - wikimedia - TriplyDB

HIF1A reduces acute lung injury by optimizing carbohydrate metabolism in the alveolar epithelium

HIF1A reduces acute lung injury by optimizing carbohydrate metabolism in the alveolar epithelium

http://www.wikidata.org/entity/Q27316863

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Hypoxia-inducible factors as molecular targets for liver diseases Circadian molecular clock in lung pathophysiology The acute respiratory distress syndrome: from mechanism to translation Potent and selective chemical probe of hypoxic signalling downstream of HIF-α hydroxylation via VHL inhibition HIF-1α is required for disturbed flow-induced metabolic reprogramming in human and porcine vascular endothelium Gas Exchange Disturbances Regulate Alveolar Fluid Clearance during Acute Lung Injury.Cell-cell interactions and bronchoconstrictor eicosanoid reduction with inhaled carbon monoxide and resolvin D1.Targeting hypoxia signalling for the treatment of ischaemic and inflammatory diseases Hypoxia-induced deoxycytidine kinase contributes to epithelial proliferation in pulmonary fibrosis.Purinergic signaling on leukocytes infiltrating the LPS-injured lung Alveolar Epithelial A2B Adenosine Receptors in Pulmonary Protection during Acute Lung Injury.In Vivo Effects of Mesenchymal Stromal Cells in Two Patients With Severe Acute Respiratory Distress Syndrome.Editorial: Health Impact and Management of a Disrupted Circadian Rhythm and Sleep in Critical Illnesses Hypoxia signaling during acute lung injury.Altered Hypoxic-Adenosine Axis and Metabolism in Group III Pulmonary Hypertension Hypoxia and inflammation are two sides of the same coin Immunologic Consequences of Hypoxia during Critical Illness.Identification of hypoxia-inducible factor HIF-1A as transcriptional regulator of the A2B adenosine receptor during acute lung injury.The regulation of pulmonary inflammation by the hypoxia-inducible factor-hydroxylase oxygen-sensing pathway.The Hypoxia-Adenosine Link during Inflammation.Recent Advances in Developing Inhibitors for Hypoxia-Inducible Factor Prolyl Hydroxylases and Their Therapeutic Implications.Hypoxia and the hypoxia-regulated transcription factor HIF-1α suppress the host defence of airway epithelial cells.DNA Repair Interacts with Autophagy To Regulate Inflammatory Responses to Pulmonary Hyperoxia.Inhibition of Prolyl Hydroxylase Attenuates Fas Ligand-Induced Apoptosis and Lung Injury in Mice.Cyclic mechanical stretch down-regulates cathelicidin antimicrobial peptide expression and activates a pro-inflammatory response in human bronchial epithelial cells.Hydroxylation-independent HIF-1α stabilization through PKA: A new paradigm for hypoxia signaling.New Insights into Protein Hydroxylation and Its Important Role in Human Diseases.Postinjury Inflammation and Organ Dysfunction.Hypoxia-inducible factors: a central link between inflammation and cancer.Atelectasis causes alveolar hypoxia-induced inflammation during uneven mechanical ventilation in rats Aberrant expression and activity of histone deacetylases in sporadic idiopathic pulmonary fibrosis.Transforming high risk to high yield Hypoxia-Inducible Factor 1α Signaling Promotes Repair of the Alveolar Epithelium after Acute Lung Injury.Targeting Hypoxia Signaling for Perioperative Organ Injury.HIF1A up-regulates the ADORA2B receptor on alternatively activated macrophages and contributes to pulmonary fibrosis.Capturing the multifactorial nature of ARDS - "Two-hit" approach to model murine acute lung injury.Lung Metabolism and Inflammation during Mechanical Ventilation; An Imaging Approach.

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HIF1A reduces acute lung injury by optimizing carbohydrate metabolism in the alveolar epithelium

http://www.wikidata.org/entity/Q27316863

description

2013 nî lūn-bûn

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2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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JOURNAL.PBIO.1001665

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HIF1A reduces acute lung injur ...... ism in the alveolar epithelium

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Christoph H Borchers

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Douglas J Kominsky

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Holger K Eltzschig

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Jun Han

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Kelley Brodsky

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Thomas J Mariani

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Tobias Eckle

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C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells

Transcriptional regulation of genes encoding glycolytic enzymes by hypoxia-inducible factor 1

Adenosine and hypoxia-inducible factor signaling in intestinal injury and recovery

Coordinated adenine nucleotide phosphohydrolysis and nucleoside signaling in posthypoxic endothelium: role of ectonucleotidases and adenosine A2B receptors

Cell-permeating alpha-ketoglutarate derivatives alleviate pseudohypoxia in succinate dehydrogenase-deficient cells

Oxygenation inhibits the physiological tissue-protecting mechanism and thereby exacerbates acute inflammatory lung injury.

Crucial role for ecto-5'-nucleotidase (CD73) in vascular leakage during hypoxia.

Hypoxia-inducible factors in physiology and medicine

Hypoxia: an alarm signal during intestinal inflammation

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10.1371/JOURNAL.PBIO.1001665

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Michel Mittelbronn

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2013-09-01T00:00:00Z

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257301897

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