Pseudomonas aeruginosa suppresses host immunity by activating the DAF-2 insulin-like signaling pathway in Caenorhabditis elegans - Wikidata - wikimedia - TriplyDB

Pseudomonas aeruginosa suppresses host immunity by activating the DAF-2 insulin-like signaling pathway in Caenorhabditis elegans

Pseudomonas aeruginosa suppresses host immunity by activating the DAF-2 insulin-like signaling pathway in Caenorhabditis elegans

http://www.wikidata.org/entity/Q27318409

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Evolution of host innate defence: insights from Caenorhabditis elegans and primitive invertebrates Gamma-linolenic and stearidonic acids are required for basal immunity in Caenorhabditis elegans through their effects on p38 MAP kinase activity Recovery from an acute infection in C. elegans requires the GATA transcription factor ELT-2 C. elegans maximum velocity correlates with healthspan and is maintained in worms with an insulin receptor mutation.Serotonergic chemosensory neurons modify the C. elegans immune response by regulating G-protein signaling in epithelial cells The DAF-16/FOXO transcription factor functions as a regulator of epidermal innate immunity Induction of cytoprotective pathways is central to the extension of lifespan conferred by multiple longevity pathways Genome-wide analysis of germline signaling genes regulating longevity and innate immunity in the nematode Pristionchus pacificus A two-gene balance regulates Salmonella typhimurium tolerance in the nematode Caenorhabditis elegans The role of insulin/IGF-1 signaling in the longevity of model invertebrates, C. elegans and D. melanogaster It takes nerves to fight infections: insights on neuro-immune interactions from C. elegans A role for SKN-1/Nrf in pathogen resistance and immunosenescence in Caenorhabditis elegans Genome-wide identification of Pseudomonas aeruginosa virulence-related genes using a Caenorhabditis elegans infection model HandKAchip - Hands-free killing assay on a chip.Meta-Analysis of Global Transcriptomics Suggests that Conserved Genetic Pathways are Responsible for Quercetin and Tannic Acid Mediated Longevity in C. elegans.The Nematode Caenorhabditis elegans, Stress and Aging: Identifying the Complex Interplay of Genetic Pathways Following the Treatment with Humic Substances.Alterations in gene expression in Caenorhabditis elegans associated with organophosphate pesticide intoxication and recovery.Control of intestinal bacterial proliferation in regulation of lifespan in Caenorhabditis elegans.Phenotypic covariance of longevity, immunity and stress resistance in the caenorhabditis nematodes.An evolutionarily conserved transcriptional response to viral infection in Caenorhabditis nematodes.The Natural Biotic Environment of Caenorhabditis elegans.C. elegans germline-deficient mutants respond to pathogen infection using shared and distinct mechanisms.C. elegans SWAN-1 Binds to EGL-9 and regulates HIF-1-mediated resistance to the bacterial pathogen Pseudomonas aeruginosa PAO1.Endoplasmic Reticulum Stress Caused by Lipoprotein Accumulation Suppresses Immunity against Bacterial Pathogens and Contributes to Immunosenescence A comprehensive analysis of gene expression changes provoked by bacterial and fungal infection in C. elegans.Environmentally induced changes in correlated responses to selection reveal variable pleiotropy across a complex genetic network Protist-type lysozymes of the nematode Caenorhabditis elegans contribute to resistance against pathogenic Bacillus thuringiensis A conserved PHD finger protein and endogenous RNAi modulate insulin signaling in Caenorhabditis elegans Systemic and cell intrinsic roles of Gqalpha signaling in the regulation of innate immunity, oxidative stress, and longevity in Caenorhabditis elegans.Selenite enhances immune response against Pseudomonas aeruginosa PA14 via SKN-1 in Caenorhabditis elegans Autophagy protects C. elegans against necrosis during Pseudomonas aeruginosa infection.Activated and inactivated immune responses in Caenorhabditis elegans against Photorhabdus luminescens TT01 Model systems to the rescue: The relationship between aging and innate immunity Antinematode activity of Violacein and the role of the insulin/IGF-1 pathway in controlling violacein sensitivity in Caenorhabditis elegans Conditioning protects C. elegans from lethal effects of enteropathogenic E. coli by activating genes that regulate lifespan and innate immunity.Commensals, probiotics and pathogens in the Caenorhabditis elegans model.Role of the nervous system in the control of proteostasis during innate immune activation: insights from C. elegans Identification of virulence properties in Salmonella Typhimurium DT104 using Caenorhabditis elegans Anti-aging effect of polysaccharide from Bletilla striata on nematode Caenorhabditis elegans Plasmodium falciparum suppresses the host immune response by inducing the synthesis of insulin-like peptides (ILPs) in the mosquito Anopheles stephensi

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Pseudomonas aeruginosa suppresses host immunity by activating the DAF-2 insulin-like signaling pathway in Caenorhabditis elegans

http://www.wikidata.org/entity/Q27318409

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2008 nî lūn-bûn

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2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2008 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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Pseudomonas aeruginosa suppres ...... hway in Caenorhabditis elegans

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Pseudomonas aeruginosa suppres ...... hway in Caenorhabditis elegans

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Pseudomonas aeruginosa suppres ...... hway in Caenorhabditis elegans

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Pseudomonas aeruginosa suppres ...... hway in Caenorhabditis elegans

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JOURNAL.PPAT.1000175

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Pseudomonas aeruginosa suppres ...... hway in Caenorhabditis elegans

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Eric A Evans

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Man-Wah Tan

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Trupti Kawli

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P2860

C. elegans SGK-1 is the critical component in the Akt/PKB kinase complex to control stress response and life span

Identification of the differential distribution patterns of mRNAs and consensus binding sequences for mouse DAF-16 homologues

The genetics of Caenorhabditis elegans

Regulation of DAF-2 receptor signaling by human insulin and ins-1, a member of the unusually large and diverse C. elegans insulin gene family

A PDK1 homolog is necessary and sufficient to transduce AGE-1 PI3 kinase signals that regulate diapause in Caenorhabditis elegans

Plants and animals share functionally common bacterial virulence factors

Use of model plant hosts to identify Pseudomonas aeruginosa virulence factors

Use of the Galleria mellonella caterpillar as a model host to study the role of the type III secretion system in Pseudomonas aeruginosa pathogenesis

p38 MAPK regulates expression of immune response genes and contributes to longevity in C. elegans

Innate immune recognition

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e1000175

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scholarly article

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2590842

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10.1371/JOURNAL.PPAT.1000175

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10

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18927620

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Pseudomonas aeruginosa

Caenorhabditis elegans

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2568960

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