Heterogeneities in Nanog Expression Drive Stable Commitment to Pluripotency in the Mouse Blastocyst
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Pluripotency Factors on Their Lineage MoveWnt/ß-catenin signalling and the dynamics of fate decisions in early mouse embryos and embryonic stem (ES) cellsMultiple cell and population-level interactions with mouse embryonic stem cell heterogeneity.Single cells get together: High-resolution approaches to study the dynamics of early mouse developmentThe many faces of Pluripotency: in vitro adaptations of a continuum of in vivo states.Four simple rules that are sufficient to generate the mammalian blastocystCell signaling and transcription factors regulating cell fate during formation of the mouse blastocyst.Inverted light-sheet microscope for imaging mouse pre-implantation development.Quantitative Analysis of Protein Expression to Study Lineage Specification in Mouse Preimplantation Embryos.Venus trap in the mouse embryo reveals distinct molecular dynamics underlying specification of first embryonic lineages.A Dynamic Role of TBX3 in the Pluripotency Circuitry.Cell Fate Specification Based on Tristability in the Inner Cell Mass of Mouse Blastocysts.Polycomb enables primitive endoderm lineage priming in embryonic stem cellsAsynchronous fate decisions by single cells collectively ensure consistent lineage composition in the mouse blastocyst.Symmetry breaking in development and stochastic gene expression.Multiple phases in regulation of Nanog expression during pre-implantation development.Signaling pathways in mammalian preimplantation development: Linking cellular phenotypes to lineage decisions.Nanog Dynamics in Mouse Embryonic Stem Cells: Results from Systems Biology Approaches.Nanog Fluctuations in Embryonic Stem Cells Highlight the Problem of Measurement in Cell Biology.A multiscale model of early cell lineage specification including cell divisionICM conversion to epiblast by FGF/ERK inhibition is limited in time and requires transcription and protein degradation.Gigantol Suppresses Cancer Stem Cell-Like Phenotypes in Lung Cancer Cells.FGF/MAPK signaling sets the switching threshold of a bistable circuit controlling cell fate decisions in embryonic stem cells.Plasticity of the inner cell mass in mouse blastocyst is restricted by the activity of FGF/MAPK pathway.Suppression of ERK signalling abolishes primitive endoderm formation but does not promote pluripotency in rabbit embryo.Distinct Requirements for FGFR1 and FGFR2 in Primitive Endoderm Development and Exit from Pluripotency.Nanog Expression in Embryonic Stem Cells - An Ideal Model System to Dissect Enhancer Function.Dynamic changes in Sox2 spatio-temporal expression promote the second cell fate decision through Fgf4/Fgfr2 signalling in preimplantation mouse embryos.
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P2860
Heterogeneities in Nanog Expression Drive Stable Commitment to Pluripotency in the Mouse Blastocyst
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Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
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Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
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Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
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Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
@ast
Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
@en
Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
@nl
prefLabel
Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
@ast
Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
@en
Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
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P2860
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Heterogeneities in Nanog Expre ...... otency in the Mouse Blastocyst
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P2093
Minjung Kang
Panagiotis Xenopoulos
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P304
P3181
P356
10.1016/J.CELREP.2015.02.010
P577
2015-03-05T00:00:00Z