The mitotic kinesin KIF11 is a driver of invasion, proliferation, and self-renewal in glioblastoma.
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Microtubule plus-end tracking proteins in neuronal developmentChromosomal Instability Affects the Tumorigenicity of Glioblastoma Tumor-Initiating Cells.Post-translational Modifications of OLIG2 Regulate Glioma Invasion through the TGF-β PathwayMiR-146b-5p overexpression attenuates stemness and radioresistance of glioma stem cells by targeting HuR/lincRNA-p21/β-catenin pathway.TGFβ-Responsive HMOX1 Expression Is Associated with Stemness and Invasion in Glioblastoma Multiforme.KIF15 promotes pancreatic cancer proliferation via the MEK-ERK signalling pathway.Overexpression of a novel candidate oncogene KIF14 correlates with tumor progression and poor prognosis in prostate cancer.Identification of candidate biomarkers and analysis of prognostic values in ovarian cancer by integrated bioinformatics analysis.ZNF131 suppresses centrosome fragmentation in glioblastoma stem-like cells through regulation of HAUS5.Syntaphilin controls a mitochondrial rheostat for proliferation-motility decisions in cancer.Molecular and Genetic Determinants of Glioma Cell Invasion.Src family kinase phosphorylation of the motor domain of the human kinesin-5, Eg5.Characterization of KIF11 as a novel prognostic biomarker and therapeutic target for oral cancer.The kinesin Eg5 inhibitor K858 induces apoptosis and reverses the malignant invasive phenotype in human glioblastoma cells.Immunologic and gene expression profiles of spontaneous canine oligodendrogliomas.A Large-Scale RNAi Screen Identifies SGK1 as a Key Survival Kinase for GBM Stem Cells.Developmentally regulated signaling pathways in glioma invasion.A posttranslational modification of the mitotic kinesin Eg5 that enhances its mechanochemical coupling and alters its mitotic function.A New Way to Treat Brain Tumors: Targeting Proteins Coded by Microcephaly Genes?: Brain tumors and microcephaly arise from opposing derangements regulating progenitor growth. Drivers of microcephaly could be attractive brain tumor targets.
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P2860
The mitotic kinesin KIF11 is a driver of invasion, proliferation, and self-renewal in glioblastoma.
description
2015 nî lūn-bûn
@nan
2015 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
The mitotic kinesin KIF11 is a ...... d self-renewal in glioblastoma
@nl
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@ast
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@en
type
label
The mitotic kinesin KIF11 is a ...... d self-renewal in glioblastoma
@nl
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@ast
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@en
prefLabel
The mitotic kinesin KIF11 is a ...... d self-renewal in glioblastoma
@nl
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@ast
The mitotic kinesin KIF11 is a ...... self-renewal in glioblastoma.
@en
P2093
P2860
P3181
P1476
The mitotic kinesin KIF11 is a ...... d self-renewal in glioblastoma
@en
P2093
Amit Vasanji
Amy C Burrows
Cathleen Chang
Craig Horbinski
James F Crish
Jennifer Major
Jeremy N Rich
John Prokop
Matthew K Summers
Monica Venere
P2860
P304
P3181
P356
10.1126/SCITRANSLMED.AAC6762
P407
P577
2015-09-01T00:00:00Z