The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction - Wikidata - wikimedia - TriplyDB

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

http://www.wikidata.org/entity/Q27334692

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Diabetic retinopathy: current understanding, mechanisms, and treatment strategies Emerging roles of RNA-binding proteins in diabetes and their therapeutic potential in diabetic complications.Deletion of the Akt/mTORC1 Repressor REDD1 Prevents Visual Dysfunction in a Rodent Model of Type 1 Diabetes.Activation of the Stress Response Kinase JNK (c-Jun N-terminal Kinase) Attenuates Insulin Action in Retina through a p70S6K1-dependent Mechanism.Cross-Dysregulation of -GlcNAcylation and PI3K/AKT/mTOR Axis in Human Chronic Diseases

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

http://www.wikidata.org/entity/Q27334692

description

2016 nî lūn-bûn

@nan

2016 թուականի Մարտին հրատարակուած գիտական յօդուած

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2016 թվականի մարտին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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type

label

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

@en

The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

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Investigative Ophthalmology & Visual Science

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The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction

@en

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Alistair J Barber

http://www.w3.org/2001/XMLSchema#string

Chen Yang

http://www.w3.org/2001/XMLSchema#string

Leonard S Jefferson

http://www.w3.org/2001/XMLSchema#string

Maria L Mihailescu

http://www.w3.org/2001/XMLSchema#string

Michael D Dennis

http://www.w3.org/2001/XMLSchema#string

Scot R Kimball

http://www.w3.org/2001/XMLSchema#string

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Phosphorylation and inactivation of glycogen synthase kinase 3 by protein kinase A

Downregulation of beta-catenin by human Axin and its association with the APC tumor suppressor, beta-catenin and GSK3 beta

Translational homeostasis via the mRNA cap-binding protein, eIF4E

Convergence of multiple signaling cascades at glycogen synthase kinase 3: Edg receptor-mediated phosphorylation and inactivation by lysophosphatidic acid through a protein kinase C-dependent intracellular pathway.

Multiple mechanisms control phosphorylation of PHAS-I in five (S/T)P sites that govern translational repression

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Control of beta-catenin phosphorylation/degradation by a dual-kinase mechanism

Function and regulation of cullin-RING ubiquitin ligases

PEST sequences and regulation by proteolysis

A tissue-specific atlas of mouse protein phosphorylation and expression

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1327-1337

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scholarly article

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10.1167/IOVS.15-18719

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3

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57

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William P Miller

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2016-03-01T00:00:00Z

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26998719

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4811182

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