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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

http://www.wikidata.org/entity/Q27481041

about

Forkhead box transcription factor regulation and lipid accumulation by hepatitis C virus.Association of lipid droplet and hepatitis C virus proteins: insights for virus replication.

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

http://www.wikidata.org/entity/Q27481041

description

2007 nî lūn-bûn

@nan

2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

name

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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type

label

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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prefLabel

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

@ast

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

@en

Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

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GUT.2006.118422

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Fatty liver in chronic hepatitis C infection: unravelling the mechanisms

@en

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Emma Paulon

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Jude A Oben

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P2860

Hepatitis C virus nonstructural proteins inhibit apolipoprotein B100 secretion

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

Insulin resistance is a cause of steatosis and fibrosis progression in chronic hepatitis C

Hepatitis C virus core protein shows a cytoplasmic localization and associates to cellular lipid storage droplets

The genotype 3-specific hepatitis C virus core protein residue phenylalanine 164 increases steatosis in an in vitro cellular model

Suppressor of cytokine signaling 1 (SOCS-1) and SOCS-3 cause insulin resistance through inhibition of tyrosine phosphorylation of insulin receptor substrate proteins by discrete mechanisms

Sterol regulatory element-binding proteins (SREBPs): transcriptional regulators of lipid synthetic genes

Cellular mechanisms of insulin resistance

Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein

Intrahepatic gene expression during chronic hepatitis C virus infection in chimpanzees.

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1186-8

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scholarly article

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10.1136/GUT.2006.118422

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9

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2007-09-01T00:00:00Z

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17698864

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