Protective immune responses against West Nile virus are primed by distinct complement activation pathways - Wikidata - wikimedia - TriplyDB

Protective immune responses against West Nile virus are primed by distinct complement activation pathways

Protective immune responses against West Nile virus are primed by distinct complement activation pathways

http://www.wikidata.org/entity/Q27482651

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Transcriptomic profile of host response in Japanese encephalitis virus infection The enhancement of arbovirus transmission and disease by mosquito saliva is associated with modulation of the host immune response Complement contributes to inflammatory tissue destruction in a mouse model of Ross River virus-induced disease Borrelia burgdorferi binding of host complement regulator factor H is not required for efficient mammalian infection The interferon-stimulated gene IFITM3 restricts West Nile virus infection and pathogenesis The Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific Manner West Nile Virus Infection in the Central Nervous System West Nile Virus: biology, transmission, and human infection Mechanism of West Nile virus neuroinvasion: a critical appraisal Age-Dependent Cell Trafficking Defects in Draining Lymph Nodes Impair Adaptive Immunity and Control of West Nile Virus Infection Pathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral Evasion West Nile virus nonstructural protein NS1 inhibits complement activation by binding the regulatory protein factor H CD4+ T-Cell Responses Are Required for Clearance of West Nile Virus from the Central Nervous System Flavivirus nonstructural protein NS1: Complementary surprises Comparison of the inhibitory effects of interferon alfacon-1 and ribavirin on yellow fever virus infection in a hamster model Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis CD40-CD40 Ligand Interactions Promote Trafficking of CD8+ T Cells into the Brain and Protection against West Nile Virus Encephalitis Induction of Epitope-Specific Neutralizing Antibodies against West Nile Virus Complement Protein C1q Inhibits Antibody-Dependent Enhancement of Flavivirus Infection in an IgG Subclass-Specific Manner Yellow fever vaccine induces integrated multilineage and polyfunctional immune responses The structural immunology of antibody protection against West Nile virus Molecular mechanisms of antibody-mediated neutralisation of flavivirus infection Complement modulates pathogenesis and antibody-dependent neutralization of West Nile virus infection through a C5-independent mechanism Progress on the development of therapeutics against West Nile virus Complement and its role in protection and pathogenesis of flavivirus infections Complement Protein C1q Reduces the Stoichiometric Threshold for Antibody-Mediated Neutralization of West Nile Virus Interferon regulatory factor-1 (IRF-1) shapes both innate and CD8(+) T cell immune responses against West Nile virus infection The interferon-inducible gene viperin restricts West Nile virus pathogenesis The Complement System in Flavivirus Infections Systems biology approach predicts immunogenicity of the yellow fever vaccine in humans Synergy between the classical and alternative pathways of complement is essential for conferring effective protection against the pandemic influenza A(H1N1) 2009 virus infection.Complex interactions with several arms of the complement system dictate innate and humoral immunity to adenoviral vectors.Surviving mousepox infection requires the complement system A complement-microglial axis drives synapse loss during virus-induced memory impairment.Complement as an endogenous adjuvant for dendritic cell-mediated induction of retrovirus-specific CTLs.Requirement for complement in antibody responses is not explained by the classic pathway activator IgM.Role and mechanism of action of complement in regulating T cell immunity.Direct complement restriction of flavivirus infection requires glycan recognition by mannose-binding lectin.Complement and viral pathogenesis Elucidating the role of the complement control protein in monkeypox pathogenicity

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P2860

Protective immune responses against West Nile virus are primed by distinct complement activation pathways

http://www.wikidata.org/entity/Q27482651

description

2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006年の論文

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Protective immune responses ag ...... complement activation pathways

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P2860

Rapid detection of west nile virus from human clinical specimens, field-collected mosquitoes, and avian samples by a TaqMan reverse transcriptase-PCR assay

Chemokine receptor CCR5 promotes leukocyte trafficking to the brain and survival in West Nile virus infection

Antibody-dependent enhancement of Ebola virus infection

Complement activation is required for induction of a protective antibody response against West Nile virus infection

Neuronal CXCL10 directs CD8+ T-cell recruitment and control of West Nile virus encephalitis.

Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survival

West Nile Virus Discriminates between DC-SIGN and DC-SIGNR for Cellular Attachment and Infection

B cells and antibody play critical roles in the immediate defense of disseminated infection by West Nile encephalitis virus.

Development of a humanized monoclonal antibody with therapeutic potential against West Nile virus

Complement receptor mediates enhanced flavivirus replication in macrophages

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1371-81

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10.1084/JEM.20052388

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5

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203

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Michael S. Diamond

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7130790

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16651386

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West Nile virus

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2121216

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