about
The sodium leak channel, NALCN, in health and diseaseThe long non-coding RNA NEAT1 is responsive to neuronal activity and is associated with hyperexcitability statesGenetic determinants of common epilepsies: a meta-analysis of genome-wide association studiesInterference of TRPV1 function altered the susceptibility of PTZ-induced seizuresDe novo loss- or gain-of-function mutations in KCNA2 cause epileptic encephalopathyHCN channels enhance spike phase coherence and regulate the phase of spikes and LFPs in the theta-frequency range.Long-term Continuous EEG Monitoring in Small Rodent Models of Human Disease Using the Epoch Wireless Transmitter System.Dual Therapeutic Effects of C-10068, a Dextromethorphan Derivative, Against Post-Traumatic Nonconvulsive Seizures and Neuroinflammation in a Rat Model of Penetrating Ballistic-Like Brain Injury.Gene Mutation Analysis in 253 Chinese Children with Unexplained Epilepsy and Intellectual/Developmental Disabilities.Influence of Genetic Variants of the N-Methyl-D-Aspartate Receptor on Emotion and Social Behavior in AdolescentsPast and present definitions of epileptogenesis and its biomarkers.Genetic biomarkers in epilepsy.GABAergic Synchronization in Epilepsy.Potassium Channels in Epilepsy.Multiscale modeling in the clinic: diseases of the brain and nervous system.The role of genetic testing in epilepsy diagnosis and management.Mutations in the sodium channel gene SCN2A cause neonatal epilepsy with late-onset episodic ataxia.Impaired neuronal operation through aberrant intrinsic plasticity in epilepsy.A novel de novo mutation of SCN8A (Nav1.6) with enhanced channel activation in a child with epileptic encephalopathy.Clinical spectrum and genotype-phenotype associations of KCNA2-related encephalopathies.High-conductance states and A-type K+ channels are potential regulators of the conductance-current balance triggered by HCN channels.Why do some brains seize? Molecular, cellular and network mechanisms.Effects of TRPV1 on the hippocampal synaptic plasticity in the epileptic rat brain.The hyperpolarization-activated cyclic nucleotide-gated (HCN) channels contain multiple S-palmitoylation sites.Genetics: Complex expressions.Functional variants in HCN4 and CACNA1H may contribute to genetic generalized epilepsy.Synaptic DisordersA Caenorhabditis elegans assay of seizure-like activity optimised for identifying antiepileptic drugs and their mechanisms of action
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Ion channels in genetic and acquired forms of epilepsy.
@ast
Ion channels in genetic and acquired forms of epilepsy.
@en
Ion channels in genetic and acquired forms of epilepsy.
@nl
type
label
Ion channels in genetic and acquired forms of epilepsy.
@ast
Ion channels in genetic and acquired forms of epilepsy.
@en
Ion channels in genetic and acquired forms of epilepsy.
@nl
prefLabel
Ion channels in genetic and acquired forms of epilepsy.
@ast
Ion channels in genetic and acquired forms of epilepsy.
@en
Ion channels in genetic and acquired forms of epilepsy.
@nl
P2093
P2860
P1476
Ion channels in genetic and acquired forms of epilepsy.
@en
P2093
P2860
P304
P356
10.1113/JPHYSIOL.2012.240606
P407
P577
2013-02-15T00:00:00Z