KRAS mutations as an independent prognostic factor in patients with advanced colorectal cancer treated with cetuximab.
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KRAS mutational analysis for colorectal cancer. Application: pharmacogenomicMarkers of resistance to anti-EGFR therapy in colorectal cancerMolecular origins of cancer: Molecular basis of colorectal cancerCurrent targeted therapies in the treatment of advanced colorectal cancer: a reviewAdvances of Targeted Therapy in Treatment of Unresectable Metastatic Colorectal CancerAdvances in targeted and immunobased therapies for colorectal cancer in the genomic eraBiomarkers predicting resistance to epidermal growth factor receptor-targeted therapy in metastatic colorectal cancer with wild-type KRASColorectal cancer tumour markers and biomarkers: Recent therapeutic advancesImpact of somatic mutations on patterns of metastasis in colorectal cancerPrognostic and Predictive Significance of Stromal Fibroblasts and Macrophages in Colon CancerMolecular phenotypes of colorectal cancer and potential clinical applicationsPrecision medicine in colorectal cancer: the molecular profile alters treatment strategiesK-Ras Mutations in Non-Small-Cell Lung Cancer: Prognostic and Predictive ValueRecent applications of chemosensitivity tests for colorectal cancer treatmentEGFR gene copy number as a prognostic marker in colorectal cancer patients treated with cetuximab or panitumumab: a systematic review and meta analysisTargeting tyrosine kinases in cancer: the converging roles of cytopathology and molecular pathology in the era of genomic medicinePIK3CA exon 20 mutations as a potential biomarker for resistance to anti-EGFR monoclonal antibodies in KRAS wild-type metastatic colorectal cancer: a systematic review and meta-analysisTargeting EML4-ALK driven non-small cell lung cancer (NSCLC)Colorectal Cancer Biomarkers: Where Are We Now?CIViC databaseIn situ selectivity profiling and crystal structure of SML-8-73-1, an active site inhibitor of oncogenic K-Ras G12CIdentification of clinically relevant molecular subtypes in colorectal cancer: the dawning of a new eraKRAS mutation in stage III colon cancer and clinical outcome following intergroup trial CALGB 89803Dasatinib sensitizes KRAS mutant colorectal tumors to cetuximabEGFR gene amplification is related to adverse clinical outcomes in cervical squamous cell carcinoma, making the EGFR pathway a novel therapeutic target.Receptor tyrosine kinases exert dominant control over PI3K signaling in human KRAS mutant colorectal cancers.PIK3CA mutation is associated with poor survival among patients with metastatic colorectal cancer following anti-EGFR monoclonal antibody therapy: a meta-analysis.Epidermal growth factor receptor immunohistochemistry: new opportunities in metastatic colorectal cancerQuantitative and sensitive detection of rare mutations using droplet-based microfluidicsPopulation pharmacokinetic analysis of panitumumab in patients with advanced solid tumorsA let-7 microRNA-binding site polymorphism in 3'-untranslated region of KRAS gene predicts response in wild-type KRAS patients with metastatic colorectal cancer treated with cetuximab monotherapyCetuximab response in CRC patient-derived xenografts seems predicted by an expression based RAS pathway signature1, 9-Pyrazoloanthrones downregulate HIF-1α and sensitize cancer cells to cetuximab-mediated anti-EGFR therapyMulti-purpose utility of circulating plasma DNA testing in patients with advanced cancersGene expression classification of colon cancer into molecular subtypes: characterization, validation, and prognostic valuePerformance and cost efficiency of KRAS mutation testing for metastatic colorectal cancer in routine diagnosis: the MOKAECM study, a nationwide experiencePCR-based assays versus direct sequencing for evaluating the effect of KRAS status on anti-EGFR treatment response in colorectal cancer patients: a systematic review and meta-analysisOverall survival of patients with KRAS wild-type tumor treated with FOLFOX/FORFIRI±cetuximab as the first-line treatment for metastatic colorectal cancer: A meta-analysis.Integrated genomic analysis of colorectal cancer progression reveals activation of EGFR through demethylation of the EREG promoterData for cancer comparative effectiveness research: past, present, and future potential.
P2860
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P2860
KRAS mutations as an independent prognostic factor in patients with advanced colorectal cancer treated with cetuximab.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
KRAS mutations as an independe ...... cancer treated with cetuximab.
@ast
KRAS mutations as an independe ...... cancer treated with cetuximab.
@en
KRAS mutations as an independe ...... cancer treated with cetuximab.
@nl
type
label
KRAS mutations as an independe ...... cancer treated with cetuximab.
@ast
KRAS mutations as an independe ...... cancer treated with cetuximab.
@en
KRAS mutations as an independe ...... cancer treated with cetuximab.
@nl
prefLabel
KRAS mutations as an independe ...... cancer treated with cetuximab.
@ast
KRAS mutations as an independe ...... cancer treated with cetuximab.
@en
KRAS mutations as an independe ...... cancer treated with cetuximab.
@nl
P2093
P50
P3181
P356
P1476
KRAS mutations as an independe ...... cancer treated with cetuximab
@en
P2093
Astrid Lièvre
Bruno Landi
Christophe Louvet
Delphine Le Corre
Emmanuel Buc
Fréderic Bibeau
Frédérique Penault-Llorca
Gorana Tomasic
Jean-François Emile
Marie-Danièle Diebold
P304
P3181
P356
10.1200/JCO.2007.12.5906
P407
P577
2008-01-01T00:00:00Z