CD47 knockout mice exhibit improved recovery from spinal cord injury. - Wikidata - wikimedia - TriplyDB

CD47 knockout mice exhibit improved recovery from spinal cord injury.

CD47 knockout mice exhibit improved recovery from spinal cord injury.

http://www.wikidata.org/entity/Q27863417

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Inflammogenesis of Secondary Spinal Cord Injury Interplay between Cellular and Molecular Inflammatory Mediators in Lung Cancer microRNAs in spinal cord injury: potential roles and therapeutic implications Thrombospondins as key regulators of synaptogenesis in the central nervous system Spinal Cord Injury CD36 deletion improves recovery from spinal cord injury.The absence of CD47 promotes nerve fiber growth from cultured ventral mesencephalic dopamine neurons Sildenafil improves epicenter vascular perfusion but not hindlimb functional recovery after contusive spinal cord injury in mice The matricellular protein thrombospondin-1 globally regulates cardiovascular function and responses to stress via CD47.SIRP/CD47 signaling in neurological disorders CD47 deficiency confers cell and tissue radioprotection by activation of autophagy.Degeneration of phrenic motor neurons induces long-term diaphragm deficits following mid-cervical spinal contusion in mice.Physiological Capillary Regression is not Dependent on Reducing VEGF Expression.Dorsal column sensory axons degenerate due to impaired microvascular perfusion after spinal cord injury in rats Vascular Pathology as a Potential Therapeutic Target in SCI.A murine toolbox for imaging the neurovascular unit.Deficiency in matrix metalloproteinase-2 results in long-term vascular instability and regression in the injured mouse spinal cord.Acute and chronic mu opioids differentially regulate thrombospondins 1 and 2 isoforms in astrocytes.Determination of Vascular Reactivity of Middle Cerebral Arteries from Stroke and Spinal Cord Injury Animal Models Using Pressure Myography.MicroRNA 486 is a potentially novel target for the treatment of spinal cord injury.

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P2860

CD47 knockout mice exhibit improved recovery from spinal cord injury.

http://www.wikidata.org/entity/Q27863417

description

2011 nî lūn-bûn

@nan

2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

CD47 knockout mice exhibit improved recovery from spinal cord injury.

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CD47 knockout mice exhibit improved recovery from spinal cord injury.

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CD47 knockout mice exhibit improved recovery from spinal cord injury.

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type

label

CD47 knockout mice exhibit improved recovery from spinal cord injury.

@ast

CD47 knockout mice exhibit improved recovery from spinal cord injury.

@en

CD47 knockout mice exhibit improved recovery from spinal cord injury.

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altLabel

CD47 knockout mice exhibit improved recovery from spinal cord injury

@en

prefLabel

CD47 knockout mice exhibit improved recovery from spinal cord injury.

@ast

CD47 knockout mice exhibit improved recovery from spinal cord injury.

@en

CD47 knockout mice exhibit improved recovery from spinal cord injury.

@nl

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J.NBD.2010.12.010

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Neurobiology of Disease

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CD47 knockout mice exhibit improved recovery from spinal cord injury.

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James B Hoying

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Kariena R Andres

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Mark J Gruenthal

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Richard L Benton

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Scott A Myers

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Scott R Whittemore

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Theo Hagg

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William H DeVries

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P2860

Cell-type specific adhesive interactions of skeletal myoblasts with thrombospondin-1

The structure of the macrophage signal regulatory protein alpha (SIRPalpha) inhibitory receptor reveals a binding face reminiscent of that used by T cell receptors

Paired receptor specificity explained by structures of signal regulatory proteins alone and complexed with CD47

Gene Expression Omnibus: NCBI gene expression and hybridization array data repository

Neurovascular effects of CD47 signaling: promotion of cell death, inflammation, and suppression of angiogenesis in brain endothelial cells in vitro

Integrin-associated protein is a receptor for the C-terminal domain of thrombospondin

SHPS-1 regulates integrin-mediated cytoskeletal reorganization and cell motility

ADAM8 is selectively up-regulated in endothelial cells and is associated with angiogenesis after spinal cord injury in adult mice

Signal-regulatory protein alpha-CD47 interactions are required for the transmigration of monocytes across cerebral endothelium

Integrin-associated protein (IAP)-deficient mice are less susceptible to developing Staphylococcus aureus-induced arthritis

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21-34

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2329799

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10.1016/J.NBD.2010.12.010

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21168495

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