CLB5-dependent activation of late replication origins in S. cerevisiae.
about
An overview of Cdk1-controlled targets and processesDisruption of mechanisms that prevent rereplication triggers a DNA damage responseAssembly of a complex containing Cdc45p, replication protein A, and Mcm2p at replication origins controlled by S-phase cyclin-dependent kinases and Cdc7p-Dbf4p kinaseBinding of cyclin-dependent kinases to ORC and Cdc6p regulates the chromosome replication cycleHow do Cdc7 and cyclin-dependent kinases trigger the initiation of chromosome replication in eukaryotic cells?Differential assembly of Cdc45p and DNA polymerases at early and late origins of DNA replicationAt short telomeres Tel1 directs early replication and phosphorylates Rif1.The mcm5-bob1 bypass of Cdc7p/Dbf4p in DNA replication depends on both Cdk1-independent and Cdk1-dependent steps in Saccharomyces cerevisiae.Distinct mechanisms control the stability of the related S-phase cyclins Clb5 and Clb6.The Rpd3-Sin3 histone deacetylase regulates replication timing and enables intra-S origin control in Saccharomyces cerevisiae.Evidence that the pre-mRNA splicing factor Clf1p plays a role in DNA replication in Saccharomyces cerevisiae.Reverse genetic analysis of the yeast RSC chromatin remodeler reveals a role for RSC3 and SNF5 homolog 1 in ploidy maintenanceInhibition of APCCdh1 activity by Cdh1/Acm1/Bmh1 ternary complex formation.Conversion of a replication origin to a silencer through a pathway shared by a Forkhead transcription factor and an S phase cyclin.G1-phase and B-type cyclins exclude the DNA-replication factor Mcm4 from the nucleus.TOR signaling is a determinant of cell survival in response to DNA damage.Establishment of cohesion at the pericentromere by the Ctf19 kinetochore subcomplex and the replication fork-associated factor, Csm3Genome-wide replication profiles indicate an expansive role for Rpd3L in regulating replication initiation timing or efficiency, and reveal genomic loci of Rpd3 function in Saccharomyces cerevisiae.Mutations in SID2, a novel gene in Saccharomyces cerevisiae, cause synthetic lethality with sic1 deletion and may cause a defect during S phaseMutational landscape of yeast mutator strainsATP-dependent chromatin remodeling shapes the DNA replication landscapeHierarchy of S-phase-promoting factors: yeast Dbf4-Cdc7 kinase requires prior S-phase cyclin-dependent kinase activation.DNA combing reveals intrinsic temporal disorder in the replication of yeast chromosome VI.Xic1 degradation in Xenopus egg extracts is coupled to initiation of DNA replication.H2A.Z functions to regulate progression through the cell cycle.Bayesian Orthogonal Least Squares (BOLS) algorithm for reverse engineering of gene regulatory networks.Segmental duplications arise from Pol32-dependent repair of broken forks through two alternative replication-based mechanisms.H3 k36 methylation helps determine the timing of cdc45 association with replication originsWhat influences DNA replication rate in budding yeast?Cell cycle arrest by transforming growth factor beta1 near G1/S is mediated by acute abrogation of prereplication complex activation involving an Rb-MCM interaction.Early initiation of a replication origin tethered at the nuclear peripheryThe DNA damage response pathway contributes to the stability of chromosome III derivatives lacking efficient replicators.Interaction of the S-phase cyclin Clb5 with an "RXL" docking sequence in the initiator protein Orc6 provides an origin-localized replication control switch.Alterations in DNA replication and histone levels promote histone gene amplification in Saccharomyces cerevisiae.Swe1 regulation and transcriptional control restrict the activity of mitotic cyclins toward replication proteins in Saccharomyces cerevisiae.Completion of replication map of Saccharomyces cerevisiae chromosome III.Initiation of eukaryotic DNA replication: conservative or liberal?Control of DNA rereplication via Cdc2 phosphorylation sites in the origin recognition complex.Cdc7-Dbf4 phosphorylates MCM proteins via a docking site-mediated mechanism to promote S phase progressionNuclear position leaves its mark on replication timing.
P2860
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P2860
CLB5-dependent activation of late replication origins in S. cerevisiae.
description
1998 nî lūn-bûn
@nan
1998 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
CLB5-dependent activation of late replication origins in S. cerevisiae.
@ast
CLB5-dependent activation of late replication origins in S. cerevisiae.
@en
CLB5-dependent activation of late replication origins in S. cerevisiae.
@nl
type
label
CLB5-dependent activation of late replication origins in S. cerevisiae.
@ast
CLB5-dependent activation of late replication origins in S. cerevisiae.
@en
CLB5-dependent activation of late replication origins in S. cerevisiae.
@nl
prefLabel
CLB5-dependent activation of late replication origins in S. cerevisiae.
@ast
CLB5-dependent activation of late replication origins in S. cerevisiae.
@en
CLB5-dependent activation of late replication origins in S. cerevisiae.
@nl
P2093
P1433
P1476
CLB5-dependent activation of late replication origins in S. cerevisiae
@en
P2093
B J Brewer
K L Friedman
M K Raghuraman
W L Fangman
P304
P356
10.1016/S1097-2765(00)80127-6
P407
P577
1998-08-01T00:00:00Z