Calcium-dependent permeabilization of erythrocytes by a perforin-like protein during egress of malaria parasites
about
Recent insights into apicomplexan parasite egress provide new views to a killA Plasmodium phospholipase is involved in disruption of the liver stage parasitophorous vacuole membraneImmune Escape Strategies of Malaria ParasitesHost cell remodeling by pathogens: the exomembrane system in Plasmodium-infected erythrocytesPerforin-like protein PPLP2 permeabilizes the red blood cell membrane during egress of Plasmodium falciparum gametocytesThe Apicomplexan CDC/MACPF-like pore-forming proteins.Parasitophorous vacuole poration precedes its rupture and rapid host erythrocyte cytoskeleton collapse in Plasmodium falciparum egress.The Plasmodium falciparum pseudoprotease SERA5 regulates the kinetics and efficiency of malaria parasite egress from host erythrocytes.Structure of the essential Plasmodium host cell traversal protein SPECT1.Plasmodium berghei calcium dependent protein kinase 1 is not required for host cell invasion.Calcium ions are involved in egress of Babesia bovis merozoites from bovine erythrocytes.Malaria parasite CelTOS targets the inner leaflet of cell membranes for pore-dependent disruption.Calcium signalling in malaria parasites.A central role for phosphatidic acid as a lipid mediator of regulated exocytosis in apicomplexa.Signaling Strategies of Malaria Parasite for Its Survival, Proliferation, and Infection during Erythrocytic Stage.Molecular Signaling Involved in Entry and Exit of Malaria Parasites from Host Erythrocytes.Measuring kinetic drivers of pneumolysin pore structure.Gliding motility powers invasion and egress in Apicomplexa.Structural Features of Apicomplexan Pore-Forming Proteins and Their Roles in Parasite Cell Traversal and EgressExploitation of a newly-identified entry pathway into the malaria parasite-infected erythrocyte to inhibit parasite egress.In silico analysis of calcium binding pocket of perforin like protein 1: insights into the regulation of pore formation.Calcium-Dependent Protein Kinase 5 Is Required for Release of Egress-Specific Organelles in Plasmodium falciparum.Effects of Estradiol and Progesterone-Induced Intracellular Calcium Fluxes on Toxoplasma gondii Gliding, Microneme Secretion, and Egress.Toxoplasma gondii LCAT Primarily Contributes to Tachyzoite Egress.Increased Ca uptake by erythrocytes infected with malaria parasites: Evidence for exported proteins and novel inhibitors
P2860
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P2860
Calcium-dependent permeabilization of erythrocytes by a perforin-like protein during egress of malaria parasites
description
2013 nî lūn-bûn
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2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@ast
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@en
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@nl
type
label
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@ast
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@en
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@nl
prefLabel
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@ast
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@en
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@nl
P2093
P2860
P921
P3181
P356
P1476
Calcium-dependent permeabiliza ...... ng egress of malaria parasites
@en
P2093
Saravanan Kumar
Shailja Singh
Shalini Agarwal
Swati Garg
Syed Shams Yazdani
P2860
P2888
P3181
P356
10.1038/NCOMMS2725
P407
P577
2013-01-01T00:00:00Z
P5875
P6179
1025644367