about
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's diseaseHost-microbe interactions have shaped the genetic architecture of inflammatory bowel diseaseMeta-analysis and imputation refines the association of 15q25 with smoking quantityTLE1 modifies the effects of NOD2 in the pathogenesis of Crohn's diseaseTuberous sclerosis-2 (TSC2) regulates the stability of death-associated protein kinase-1 (DAPK) through a lysosome-dependent degradation pathwayThe intermediate filament protein, vimentin, is a regulator of NOD2 activityCommon variants at five new loci associated with early-onset inflammatory bowel diseaseGenome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility lociGenome-wide association study of CNVs in 16,000 cases of eight common diseases and 3,000 shared controlsComparative genetic analysis of inflammatory bowel disease and type 1 diabetes implicates multiple loci with opposite effectsDiverse genome-wide association studies associate the IL12/IL23 pathway with Crohn DiseaseIL23R variation determines susceptibility but not disease phenotype in inflammatory bowel diseaseAssociation scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variantsGenome-wide association study of ulcerative colitis identifies three new susceptibility loci, including the HNF4A regionDanish cohort of monozygotic inflammatory bowel disease twins: Clinical characteristics and inflammatory activity
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description
hulumtues
@sq
onderzoeker
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researcher
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ricercatore
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հետազոտող
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name
Jack Satsangi
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Jack Satsangi
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Jack Satsangi
@es
Jack Satsangi
@sl
type
label
Jack Satsangi
@ast
Jack Satsangi
@en
Jack Satsangi
@es
Jack Satsangi
@sl
prefLabel
Jack Satsangi
@ast
Jack Satsangi
@en
Jack Satsangi
@es
Jack Satsangi
@sl
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