ErbB2 signaling at the crossing between heart failure and cancer - Wikidata - wikimedia - TriplyDB

ErbB2 signaling at the crossing between heart failure and cancer

ErbB2 signaling at the crossing between heart failure and cancer

http://www.wikidata.org/entity/Q28075244

about

The antineoplastic drug, trastuzumab, dysregulates metabolism in iPSC-derived cardiomyocytes.Cardiac Regeneration: Lessons From Development.Investigation of the role of DNA methylation in the expression of ERBB2 in human myocardium.Cardiovascular toxic effects of targeted cancer therapy.Cardiac Remodeling: Endothelial Cells Have More to Say Than Just NO.

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ErbB2 signaling at the crossing between heart failure and cancer

http://www.wikidata.org/entity/Q28075244

description

2016 nî lūn-bûn

@nan

2016 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2016年の論文

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2016年論文

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2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

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2016年論文

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2016年论文

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name

ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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type

label

ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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prefLabel

ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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ErbB2 signaling at the crossing between heart failure and cancer

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Basic Research in Cardiology

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ErbB2 signaling at the crossing between heart failure and cancer

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Zarha Vermeulen

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Creative Commons Attribution 4.0 International

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Neuregulins promote survival and growth of cardiac myocytes. Persistence of ErbB2 and ErbB4 expression in neonatal and adult ventricular myocytes

Ligand-independent HER2/HER3/PI3K complex is disrupted by trastuzumab and is effectively inhibited by the PI3K inhibitor GDC-0941

A phase II study of afatinib (BIBW 2992), an irreversible ErbB family blocker, in patients with HER2-positive metastatic breast cancer progressing after trastuzumab

Escape from HER-family tyrosine kinase inhibitor therapy by the kinase-inactive HER3

Phosphotyrosine interactome of the ErbB-receptor kinase family

The ErbB2/ErbB3 heterodimer functions as an oncogenic unit: ErbB2 requires ErbB3 to drive breast tumor cell proliferation

Intermolecular biparatopic trapping of ErbB2 prevents compensatory activation of PI3K/AKT via RAS-p110 crosstalk.

Use of chemotherapy plus a monoclonal antibody against HER2 for metastatic breast cancer that overexpresses HER2.

Neratinib, an irreversible ErbB receptor tyrosine kinase inhibitor, in patients with advanced ErbB2-positive breast cancer.

Trastuzumab emtansine for HER2-positive advanced breast cancer.

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10.1007/S00395-016-0576-Z

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Gilles W De Keulenaer

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cell surface receptor

membrane proteins

proto-oncogene proteins

tumor biomarkers

neoplasm proteins

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