ATG16L1: A multifunctional susceptibility factor in Crohn disease - Wikidata - wikimedia - TriplyDB

ATG16L1: A multifunctional susceptibility factor in Crohn disease

ATG16L1: A multifunctional susceptibility factor in Crohn disease

http://www.wikidata.org/entity/Q28082887

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Genetic Influences on the Development of Fibrosis in Crohn's Disease Roles of Autophagy and Autophagy-Related Proteins in Antifungal Immunity Homeostasis of the gut barrier and potential biomarkers.Amyloid formation: functional friend or fearful foe?Cerebral magnetic resonance imaging in quiescent Crohn's disease patients with fatigue Analysis of the human monocyte-derived macrophage transcriptome and response to lipopolysaccharide provides new insights into genetic aetiology of inflammatory bowel disease.C-reactive protein levels in the perioperative period as a predictive marker of endoscopic recurrence after ileo-colonic resection for Crohn's disease.Variants in autophagy-related genes and clinical characteristics in melanoma: a population-based study Diet and microbiota in inflammatory bowel disease: The gut in disharmony.NOD2 induces autophagy to control AIEC bacteria infectiveness in intestinal epithelial cells.MicroRNAs: how many in inflammatory bowel disease?A functional ATG16L1 (T300A) variant is associated with necrotizing enterocolitis in premature infants.Role of MiRNAs in Inflammatory Bowel Disease.Impact of T300A Variant of ATG16L1 on Antibacterial Response, Risk of Culture Positive Infections, and Clinical Course of Crohn's Disease.Innate Immunity to Intracellular Pathogens: Balancing Microbial Elimination and Inflammation.Danger of frustrated sensors: Role of Toll-like receptors and NOD-like receptors in aseptic and septic inflammations around total hip replacements.Trichothecenes: immunomodulatory effects, mechanisms, and anti-cancer potential.Establishment of S100A8 Transgenic Rats to Understand Innate Property of S100A8 and Its Immunological Role.Association between ATG16L1 gene polymorphism and the risk of Crohn's disease.Mechanisms and function of autophagy in intestinal disease.A polymorphism upstream MIR1279 gene is associated with pericarditis development in Systemic Lupus Erythematosus and contributes to definition of a genetic risk profile for this complication.The Thr300Ala variant of ATG16L1 is associated with decreased risk of brain metastasis in patients with non-small cell lung cancer.NOD2 and inflammation: current insights.From blood to lung tissue: effect of cigarette smoke on DNA methylation and lung function

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

http://www.wikidata.org/entity/Q28082887

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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type

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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prefLabel

ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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ATG16L1: A multifunctional susceptibility factor in Crohn disease

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Kris Nys

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Mette Ammitzboell

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P2860

Protection against fatal Sindbis virus encephalitis by beclin, a novel Bcl-2-interacting protein

A Nonsynonymous SNP in ATG16L1 Predisposes to Ileal Crohn’s Disease and Is Independent of CARD15 and IBD5

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Bif-1 interacts with Beclin 1 through UVRAG and regulates autophagy and tumorigenesis

The hairpin-type tail-anchored SNARE syntaxin 17 targets to autophagosomes for fusion with endosomes/lysosomes

Ambra1 regulates autophagy and development of the nervous system

Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

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585-594

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scholarly article

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Ole Haagen Nielsen

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275358208

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