The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
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Can physical exercise in old age improve memory and hippocampal function?Molecular Pathological Classification of Neurodegenerative Diseases: Turning towards Precision Medicine3D Visualization of the Temporal and Spatial Spread of Tau Pathology Reveals Extensive Sites of Tau Accumulation Associated with Neuronal Loss and Recognition Memory Deficit in Aged Tau Transgenic MiceTowards a systems approach for chronic diseases, based on health state modeling.Association of Amyloid Pathology With Myelin Alteration in Preclinical Alzheimer Disease.Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.Chronic intermittent hypoxia induces oxidative stress and inflammation in brain regions associated with early-stage neurodegeneration.Microbes and Alzheimer's Disease.Shared genetic risk between corticobasal degeneration, progressive supranuclear palsy, and frontotemporal dementia.Active zone proteins are transported via distinct mechanisms regulated by Par-1 kinase.An In Vivo (11)C-(R)-PK11195 PET and In Vitro Pathology Study of Microglia Activation in Creutzfeldt-Jakob Disease.The Locus Coeruleus: Essential for Maintaining Cognitive Function and the Aging Brain.In Vivo Detection of Amyloid Plaques by Gadolinium-Stained MRI Can Be Used to Demonstrate the Efficacy of an Anti-amyloid ImmunotherapyThe incubation period of Alzheimer's disease and the timing of tau versus amyloid misfolding and spreading within the brain.Dura mater is a potential source of Aβ seeds.Identification of learning-induced changes in protein networks in the hippocampi of a mouse model of Alzheimer's disease.Changes in brain oxysterols at different stages of Alzheimer's disease: Their involvement in neuroinflammationIdentify the Atrophy of Alzheimer's Disease, Mild Cognitive Impairment and Normal Aging Using Morphometric MRI Analysis.Soluble phospho-tau from Alzheimer's disease hippocampus drives microglial degeneration.Patterns of Cortical and Subcortical Amyloid Burden across Stages of Preclinical Alzheimer's Disease.Epigenetic dysregulation of brainstem nuclei in the pathogenesis of Alzheimer's disease: looking in the correct place at the right time?Review: Sporadic Parkinson's disease: development and distribution of α-synuclein pathology.Propagation of Aß pathology: hypotheses, discoveries, and yet unresolved questions from experimental and human brain studies.Monoaminergic neuropathology in Alzheimer's disease.Potential Pathways of Abnormal Tau and α-Synuclein Dissemination in Sporadic Alzheimer's and Parkinson's Diseases.Is sporadic Alzheimer's disease a developmental disorder?Exercise as a Positive Modulator of Brain Function.Detectable Neuropsychological Differences in Early Preclinical Alzheimer's Disease: A Meta-Analysis.Low-Frequency Pulsed Electromagnetic Field Is Able to Modulate miRNAs in an Experimental Cell Model of Alzheimer's Disease.Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer's disease.Pupillary Responses as a Biomarker of Early Risk for Alzheimer's Disease.Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and Disease.Brainstem tau pathology in Alzheimer's disease is characterized by increase of three repeat tau and independent of amyloid β.Multiscale network modeling of oligodendrocytes reveals molecular components of myelin dysregulation in Alzheimer's disease.Tau-mediated Neurodegeneration and Potential Implications in Diagnosis and Treatment of Alzheimer's Disease.Sex differences in sleep apnea and comorbid neurodegenerative diseases.MicroRNA Expression in the Locus Coeruleus, Entorhinal Cortex, and Hippocampus at Early and Middle Stages of Braak Neurofibrillary Tangle Pathology.Presynaptic proteins complexin-I and complexin-II differentially influence cognitive function in early and late stages of Alzheimer's disease.Locus coeruleus at asymptomatic early and middle Braak stages of neurofibrillary tangle pathology.Structural tract alterations predict downstream tau accumulation in amyloid-positive older individuals.
P2860
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P2860
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
description
2015 nî lūn-bûn
@nan
2015 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@ast
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@en
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@nl
type
label
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@ast
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@en
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@nl
prefLabel
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@ast
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@en
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@nl
P2860
P3181
P356
P1433
P1476
The preclinical phase of the pathological process underlying sporadic Alzheimer's disease
@en
P2093
Heiko Braak
Kelly Del Tredici
P2860
P304
P3181
P356
10.1093/BRAIN/AWV236
P407
P577
2015-10-01T00:00:00Z