Mutation of the SHP-2 binding site in growth hormone (GH) receptor prolongs GH-promoted tyrosyl phosphorylation of GH receptor, JAK2, and STAT5B
about
Estrogen inhibits GH signaling by suppressing GH-induced JAK2 phosphorylation, an effect mediated by SOCS-2Interaction of the tyrosine phosphatase SHP-2 with Gab2 regulates Rho-dependent activation of the c-fos serum response element by interleukin-2Differential Role of Leptin and Adiponectin in Cardiovascular SystemAutophosphorylation of JAK2 on tyrosines 221 and 570 regulates its activitySOCS2 negatively regulates growth hormone action in vitro and in vivoSignalling cross-talk between hepatocyte nuclear factor 4alpha and growth-hormone-activated STAT5bDefinition of the cattle killer cell Ig-like receptor gene family: comparison with aurochs and human counterpartsJAK and MPL mutations in myeloid malignancies.Knock your SOCS off!GH modulates hepatic epidermal growth factor signaling in the mouse.PTPN11 mutations in Noonan syndrome: molecular spectrum, genotype-phenotype correlation, and phenotypic heterogeneityProtein tyrosine phosphatase 1B attenuates growth hormone-mediated JAK2-STAT signaling.Specific dephosphorylation of Janus Kinase 2 by protein tyrosine phosphatases.Growth hormone-induced JAK2 signaling and GH receptor down-regulation: role of GH receptor intracellular domain tyrosine residuesRac1 links integrin-mediated adhesion to the control of lactational differentiation in mammary epitheliaModulation of growth hormone receptor abundance and function: roles for the ubiquitin-proteasome system.IGF-1R modulation of acute GH-induced STAT5 signaling: role of protein tyrosine phosphatase activity.Towards identification of molecular mechanisms of short stature.PD-1 signaling in primary T cellsGenetics of human growthLeptin in inflammation and autoimmunity.Desensitization of the JAK2/STAT5 GH signaling pathway associated with increased CIS protein content in liver of pregnant mice.PTPN11 mutations in Noonan syndrome type I: detection of recurrent mutations in exons 3 and 13.The Growth Hormone Receptor: Mechanism of Receptor Activation, Cell Signaling, and Physiological Aspects.[Phenotype variability in Noonan syndrome patients with and without PTPN11 mutation].
P2860
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P2860
Mutation of the SHP-2 binding site in growth hormone (GH) receptor prolongs GH-promoted tyrosyl phosphorylation of GH receptor, JAK2, and STAT5B
description
2000 nî lūn-bûn
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2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
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2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
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name
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@ast
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@en
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@nl
type
label
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@ast
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@en
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@nl
prefLabel
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@ast
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@en
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@nl
P2093
P356
P1476
Mutation of the SHP-2 binding ...... GH receptor, JAK2, and STAT5B
@en
P2093
C Carter-Su
J Herrington
M R Stofega
N Billestrup
P304
P356
10.1210/MEND.14.9.0513
P407
P577
2000-09-01T00:00:00Z