Cav1.4alpha1 subunits can form slowly inactivating dihydropyridine-sensitive L-type Ca2+ channels lacking Ca2+-dependent inactivation
about
A CACNA1F mutation identified in an X-linked retinal disorder shifts the voltage dependence of Cav1.4 channel activation.Essential role of Ca2+-binding protein 4, a Cav1.4 channel regulator, in photoreceptor synaptic function.X linked cone-rod dystrophy, CORDX3, is caused by a mutation in the CACNA1F geneThe Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialVoltage-Gated Cav1 Channels in Disorders of Vision and HearingA critical role of CaBP4 in the cone synapse.Calcium-induced calcium release contributes to synaptic release from mouse rod photoreceptorsCharacterization of C-terminal Splice Variants of Cav1.4 Ca2+ Channels in Human RetinaThe ß subunit of voltage-gated Ca2+ channelsGenetic dissection of horizontal cell inhibitory signaling in mice in complete darkness in vivo.Location of release sites and calcium-activated chloride channels relative to calcium channels at the photoreceptor ribbon synapse.The nob2 mouse, a null mutation in Cacna1f: anatomical and functional abnormalities in the outer retina and their consequences on ganglion cell visual responsesCav1.4 IT mouse as model for vision impairment in human congenital stationary night blindness type 2.Isoform-specific regulation of mood behavior and pancreatic beta cell and cardiovascular function by L-type Ca 2+ channelsDissecting the functional role of different isoforms of the L-type Ca2+ channel.Visual threshold is set by linear and nonlinear mechanisms in the retina that mitigate noise: how neural circuits in the retina improve the signal-to-noise ratio of the single-photon responseSlowed N-type calcium channel (CaV2.2) deactivation by the cyclin-dependent kinase inhibitor roscovitine.Cav1.4 encodes a calcium channel with low open probability and unitary conductance.Synaptic transmission at retinal ribbon synapsesRod bipolar cells and horizontal cells form displaced synaptic contacts with rods in the outer nuclear layer of the nob2 retinaIdentification of calcium channel alpha1 subunit mRNA expressed in retinal bipolar neurons.Switching off calcium-dependent inactivation in L-type calcium channels by an autoinhibitory domainCharacterization of Cav1.4 complexes (α11.4, β2, and α2δ4) in HEK293T cells and in the retinaFunctional roles of cytoplasmic loops and pore lining transmembrane helices in the voltage-dependent inactivation of HVA calcium channels.Synaptic release at mammalian bipolar cell terminalsAlternative splicing at C terminus of Ca(V)1.4 calcium channel modulates calcium-dependent inactivation, activation potential, and current density.Cav1.4 L-Type Calcium Channels Contribute to Calpain Activation in Degenerating Photoreceptors of rd1 Mice.Gain-of-function nature of Cav1.4 L-type calcium channels alters firing properties of mouse retinal ganglion cells.Kinetics of synaptic transmission at ribbon synapses of rods and cones.Transducin translocation contributes to rod survival and enhances synaptic transmission from rods to rod bipolar cellsEffect of knockout of α2δ-1 on action potentials in mouse sensory neuronsWhat can naturally occurring mutations tell us about Ca(v)1.x channel function?The molecular architecture of ribbon presynaptic terminalsMosaic synaptopathy and functional defects in Cav1.4 heterozygous mice and human carriers of CSNB2.Channelopathies in Cav1.1, Cav1.3, and Cav1.4 voltage-gated L-type Ca2+ channels.L-type Ca2+ channels in heart and brainStructural determinants of L-type channel activation in segment IIS6 revealed by a retinal disorder.Behavioural and physiological limits to vision in mammals.Mutation associated with an autosomal dominant cone-rod dystrophy CORD7 modifies RIM1-mediated modulation of voltage-dependent Ca2+ channels.Complex regulation of voltage-dependent activation and inactivation properties of retinal voltage-gated Cav1.4 L-type Ca2+ channels by Ca2+-binding protein 4 (CaBP4).
P2860
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P2860
Cav1.4alpha1 subunits can form slowly inactivating dihydropyridine-sensitive L-type Ca2+ channels lacking Ca2+-dependent inactivation
description
2003 nî lūn-bûn
@nan
2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@ast
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@en
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@nl
type
label
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@ast
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@en
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@nl
prefLabel
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@ast
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@en
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@nl
P2093
P3181
P1476
Cav1.4alpha1 subunits can form ...... ng Ca2+-dependent inactivation
@en
P2093
Alexandra Koschak
Daniel Reimer
Doris Walter
Jean-Charles Hoda
Manfred Grabner
Thomas Heinzle
P304
P3181
P356
10.1523/JNEUROSCI.23-14-06041.2003
P407
P577
2003-07-09T00:00:00Z