Differential effects of tumor necrosis factor-alpha on protein kinase C isoforms alpha and delta mediate inhibition of insulin receptor signaling

Differential effects of tumor necrosis factor-alpha on protein kinase C isoforms alpha and delta mediate inhibition of insulin receptor signaling

Item
http://www.wikidata.org/entity/Q28198256
Q28156161
sameAs
Phosphorylation of Ser357 of rat insulin receptor substrate-1 mediates adverse effects of protein kinase C-delta on insulin action in skeletal muscle cellsA Potential Role for Pro-Inflammatory Cytokines in the Development of Insulin Resistance in HorsesRegulation of insulin receptor substrate 1 pleckstrin homology domain by protein kinase C: role of serine 24 phosphorylationProtein kinase Cδ promotes proliferation and induces malignant transformation in skeletal musclePhosphorylation of adaptor protein containing pleckstrin homology domain, phosphotyrosine binding domain, and leucine zipper motif 1 (APPL1) at Ser430 mediates endoplasmic reticulum (ER) stress-induced insulin resistance in hepatocytesSpecific protein kinase C isoforms as transducers and modulators of insulin signalingRetinol as a cofactor for PKCδ-mediated impairment of insulin sensitivity in a mouse model of diet-induced obesityTrauma and hemorrhage-induced acute hepatic insulin resistance: dominant role of tumor necrosis factor-alpha.The phosphatidylethanolamine derivative diDCP-LA-PE mimics intracellular insulin signalingCeramide-activated protein phosphatase involvement in insulin resistance via Akt, serine/arginine-rich protein 40, and ribonucleic acid splicing in L6 skeletal muscle cellsPhosphorylation of IRS proteins, insulin action, and insulin resistance.Cholinergic activation suppresses palmitate-induced macrophage activation and improves acylation stimulating protein resistance in co-cultured adipocytes.Protein kinase Cδ but not PKCα is involved in insulin-induced glucose metabolism in hepatocytes.Potential beneficial mechanisms of insulin (glucose-potassium) in acute myocardial infarction.Increased glucose uptake promotes oxidative stress and PKC-delta activation in adipocytes of obese, insulin-resistant mice.Role of tumour necrosis factor alpha, but not of cyclo-oxygenase-2-derived eicosanoids, on functional and morphological indices of dystrophic progression in mdx mice: a pharmacological approach.
P2860

Differential effects of tumor necrosis factor-alpha on protein kinase C isoforms alpha and delta mediate inhibition of insulin receptor signaling

Item
http://www.wikidata.org/entity/Q28198256
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Differential effects of tumor ...... of insulin receptor signaling
@ast
Differential effects of tumor ...... of insulin receptor signaling
@en
Differential effects of tumor ...... of insulin receptor signaling
@nl
type
label
Differential effects of tumor ...... of insulin receptor signaling
@ast
Differential effects of tumor ...... of insulin receptor signaling
@en
Differential effects of tumor ...... of insulin receptor signaling
@nl
prefLabel
Differential effects of tumor ...... of insulin receptor signaling
@ast
Differential effects of tumor ...... of insulin receptor signaling
@en
Differential effects of tumor ...... of insulin receptor signaling
@nl
P1433
P1476
P2093
P304
P31
P3181
P356
P407
P433
P478
P577
P5875
P698
P921
P3181
P356
P1433
P1476
Differential effects of tumor ...... of insulin receptor signaling
@en
P2093
P304
P31
P3181
P356
10.2337/DIABETES.51.6.1921
P407
P433
P478
P577
P5875
P698
P921