Maintenance of integrated proviral gene expression requires Brm, a catalytic subunit of SWI/SNF complex
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Retroviral elements and their hosts: insertional mutagenesis in the mouse germ line.An unmethylated 3' promoter-proximal region is required for efficient transcription initiationTwo chromatin remodeling activities cooperate during activation of hormone responsive promoters.An emerging role for bromodomain-containing proteins in chromatin regulation and transcriptional control of adipogenesis.Flavonoids from each of the six structural groups reactivate BRM, a possible cofactor for the anticancer effects of flavonoidsLatency of viral expression in vivo is not related to CpG methylation in the U3 region and part of the R region of the long terminal repeat of bovine leukemia virusHigh-frequency epigenetic repression and silencing of retroviruses can be antagonized by histone deacetylase inhibitors and transcriptional activators, but uniform reactivation in cell clones is restricted by additional mechanisms.Identification of cellular proteins that maintain retroviral epigenetic silencing: evidence for an antiviral response.Functional specificities of Brm and Brg-1 Swi/Snf ATPases in the feedback regulation of hepatic bile acid biosynthesis.Two BRM promoter insertion polymorphisms increase the risk of early-stage upper aerodigestive tract cancers.The miR-199a/Brm/EGR1 axis is a determinant of anchorage-independent growth in epithelial tumor cell lines.Identifying targets for the restoration and reactivation of BRM.Double plant homeodomain (PHD) finger proteins DPF3a and -3b are required as transcriptional co-activators in SWI/SNF complex-dependent activation of NF-κB RelA/p50 heterodimer.Requiem protein links RelB/p52 and the Brm-type SWI/SNF complex in a noncanonical NF-kappaB pathway.Loss of the Brm-type SWI/SNF chromatin remodeling complex is a strong barrier to the Tat-independent transcriptional elongation of human immunodeficiency virus type 1 transcripts.The Brm gene suppressed at the post-transcriptional level in various human cell lines is inducible by transient HDAC inhibitor treatment, which exhibits antioncogenic potential.Expression of a model gene in prostate cancer cells lentivirally transduced in vitro and in vivo.SW13 cells can transition between two distinct subtypes by switching expression of BRG1 and Brm genes at the post-transcriptional level.Pharmacologic reversal of epigenetic silencing of the anticancer protein BRM: a novel targeted treatment strategy.
P2860
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P2860
Maintenance of integrated proviral gene expression requires Brm, a catalytic subunit of SWI/SNF complex
description
2002 nî lūn-bûn
@nan
2002 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@ast
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@en
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@nl
type
label
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@ast
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@en
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@nl
prefLabel
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@ast
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@en
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@nl
P2093
P2860
P356
P1476
Maintenance of integrated prov ...... tic subunit of SWI/SNF complex
@en
P2093
Akiko Watanabe
Mitsue Nishina
Nobutake Yamamichi
Taketoshi Mizutani
P2860
P304
P356
10.1074/JBC.M112421200
P407
P577
2002-05-03T00:00:00Z