S100A1 in human heart failure: lack of recovery following left ventricular assist device support
about
Cardiomyocytes, endothelial cells and cardiac fibroblasts: S100A1's triple action in cardiovascular pathophysiology.Pathophysiological mechanism and therapeutic role of S100 proteins in cardiac failure: a systematic review.Cardiac Non-myocyte Cells Show Enhanced Pharmacological Function Suggestive of Contractile Maturity in Stem Cell Derived Cardiomyocyte Microtissues.
P2860
S100A1 in human heart failure: lack of recovery following left ventricular assist device support
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
S100A1 in human heart failure: ...... tricular assist device support
@ast
S100A1 in human heart failure: ...... tricular assist device support
@en
S100A1 in human heart failure: ...... tricular assist device support
@nl
type
label
S100A1 in human heart failure: ...... tricular assist device support
@ast
S100A1 in human heart failure: ...... tricular assist device support
@en
S100A1 in human heart failure: ...... tricular assist device support
@nl
prefLabel
S100A1 in human heart failure: ...... tricular assist device support
@ast
S100A1 in human heart failure: ...... tricular assist device support
@en
S100A1 in human heart failure: ...... tricular assist device support
@nl
P2093
P2860
P3181
P1476
S100A1 in human heart failure: ...... tricular assist device support
@en
P2093
Christine S Moravec
Elizabeth Looney
Kristen Karohl
Maria Mountis
Mosi K Bennett
Sara Baicker-McKee
W H Wilson Tang
Wendy E Sweet
P2860
P3181
P356
10.1161/CIRCHEARTFAILURE.113.000849
P407
P577
2014-07-01T00:00:00Z