Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms - Wikidata - wikimedia - TriplyDB

Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms

Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms

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Celiac disease and non-celiac gluten sensitivity The potential utility of tight junction regulation in celiac disease: focus on larazotide acetate Nutritional Keys for Intestinal Barrier Modulation Viral Infection of the Central Nervous System Exacerbates Interleukin-10 Receptor Deficiency-Mediated Colitis in SJL Mice Visualization of transepithelial passage of the immunogenic 33-residue peptide from alpha-2 gliadin in gluten-sensitive macaques Leaky gut and autoimmune diseases Possible links between intestinal permeability and food processing: A potential therapeutic niche for glutamine Non-celiac gluten sensitivity: literature review Autoimmunity and the gut Role of intestinal bacteria in gliadin-induced changes in intestinal mucosa: study in germ-free rats Intestinal barrier dysfunction develops at the onset of experimental autoimmune encephalomyelitis, and can be induced by adoptive transfer of auto-reactive T cells.Restoration of impaired intestinal barrier function by the hydrolysed casein diet contributes to the prevention of type 1 diabetes in the diabetes-prone BioBreeding rat.Autism and dietary therapy: case report and review of the literature.Celiac disease and autoimmune-associated conditions.Investigation of the putative associations between dairy consumption and incidence of type 1 and type 2 diabetes.Zinc in gut-brain interaction in autism and neurological disorders.Migraine associated with gastrointestinal disorders: review of the literature and clinical implications.Do psychological factors predict symptom severity in patients with subjective food hypersensitivity?The role of IgG hypersensitivity in the pathogenesis and therapy of depressive disorders.Antibodies to the wheat storage globulin Glo-3A in children before and at diagnosis of celiac disease.Over-starvation aggravates intestinal injury and promotes bacterial and endotoxin translocation under high-altitude hypoxic environment.Multiple nonglycemic genomic loci are newly associated with blood level of glycated hemoglobin in East Asians.Increased density of tolerogenic dendritic cells in the small bowel mucosa of celiac patients.Association analysis of genetic variants in the myosin IXB gene in acute pancreatitis Naturally transmitted segmented filamentous bacteria segregate with diabetes protection in nonobese diabetic mice.Claudin-2 as a mediator of leaky gut barrier during intestinal inflammation.Diet and host-microbial crosstalk in postnatal intestinal immune homeostasis.Does the microbiota play a role in the pathogenesis of autoimmune diseases?The gut microbiota and Type 1 Diabetes.Diet and specific microbial exposure trigger features of environmental enteropathy in a novel murine model Immunopathogenesis of IBD: current state of the art.Oral Probiotic VSL#3 Prevents Autoimmune Diabetes by Modulating Microbiota and Promoting Indoleamine 2,3-Dioxygenase-Enriched Tolerogenic Intestinal Environment.Apple-Derived Pectin Modulates Gut Microbiota, Improves Gut Barrier Function, and Attenuates Metabolic Endotoxemia in Rats with Diet-Induced Obesity Does the Microbiome Cause B27-related Acute Anterior Uveitis?Large Gliadin Peptides Detected in the Pancreas of NOD and Healthy Mice following Oral Administration.On the etiology of type 1 diabetes: a new animal model signifying a decisive role for bacteria eliciting an adverse innate immunity response.Sensitization to gliadin induces moderate enteropathy and insulitis in nonobese diabetic-DQ8 mice.Pathways involved in sasang constitution from genome-wide analysis in a Korean population.Safety concerns over the use of intestinal permeation enhancers: A mini-review Faecal biomarker patterns in patients with symptoms of irritable bowel syndrome.

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Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms

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2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Tight junctions, intestinal pe ...... and type 1 diabetes paradigms

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Annals of the New York Academy of Sciences

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Tight junctions, intestinal pe ...... and type 1 diabetes paradigms

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Anna Sapone

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Jan Rozing

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Jeroen Visser

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Karen Lammers

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P2860

Structure of the human class I histocompatibility antigen, HLA-A2

Gliadin induces an increase in intestinal permeability and zonulin release by binding to the chemokine receptor CXCR3

The HLA-DQ2 gene dose effect in celiac disease is directly related to the magnitude and breadth of gluten-specific T cell responses

Myosin IXB variant increases the risk of celiac disease and points toward a primary intestinal barrier defect

Prediction of silent celiac disease at diagnosis of childhood type 1 diabetes by tissue transglutaminase autoantibodies and HLA.

Putting the pieces of the puzzle together - a series of hypotheses on the etiology and pathogenesis of type 1 diabetes.

Role of the intestinal tight junction modulator zonulin in the pathogenesis of type I diabetes in BB diabetic-prone rats.

Zonula occludens toxin structure-function analysis. Identification of the fragment biologically active on tight junctions and of the zonulin receptor binding domain.

Anatomical basis of tolerance and immunity to intestinal antigens.

Zonula occludens toxin modulates tight junctions through protein kinase C-dependent actin reorganization, in vitro

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26303176

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19538307

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type-1 diabetes

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