Evidence that one subset of anaplastic thyroid carcinomas are derived from papillary carcinomas due to BRAF and p53 mutations - Wikidata - wikimedia - TriplyDB

Evidence that one subset of anaplastic thyroid carcinomas are derived from papillary carcinomas due to BRAF and p53 mutations

Evidence that one subset of anaplastic thyroid carcinomas are derived from papillary carcinomas due to BRAF and p53 mutations

http://www.wikidata.org/entity/Q28249293

about

Gene expression profiling associated with the progression to poorly differentiated thyroid carcinomas Combined MEK and Pi3'-kinase inhibition reveals synergy in targeting thyroid cancer in vitro and in vivo.Anaplastic thyroid cancer: molecular pathogenesis and emerging therapies.Diagnosis and treatment of pancreatic metastases of a papillary thyroid carcinoma Lessons from mouse models of thyroid cancer.Anaplastic Thyroid Carcinoma: Current Treatments and Potential New Therapeutic Options with Emphasis on TfR1/CD71.The roles of the epithelial-mesenchymal transition marker PRRX1 and miR-146b-5p in papillary thyroid carcinoma progression Molecular and other novel advances in treatment of metastatic epithelial and medullary thyroid cancers Update on anaplastic thyroid carcinoma: morphological, molecular, and genetic features of the most aggressive thyroid cancer.Old and new insights in the treatment of thyroid carcinoma.mRNA expression in papillary and anaplastic thyroid carcinoma: molecular anatomy of a killing switch Increasing incidence of thyroid cancer in the Commonwealth of Pennsylvania.Expression of cancer stem cell markers and epithelial-mesenchymal transition-related factors in anaplastic thyroid carcinoma.Targeted treatment of differentiated and medullary thyroid cancer.Genetic alterations in poorly differentiated and undifferentiated thyroid carcinomas Our approach to follicular-patterned lesions of the thyroid Poorly differentiated thyroid carcinoma: an incubating entity.Modeling thyroid cancer in the mouse.Pathogenetic mechanisms in thyroid follicular-cell neoplasia.CD133-expressing thyroid cancer cells are undifferentiated, radioresistant and survive radioiodide therapy Clinical significance of preoperative detection of serum p53 antibodies and BRAF(V600E) mutation in patients with papillary thyroid carcinoma.Poorly differentiated and anaplastic thyroid cancer.Identification of potential therapeutic targets for papillary thyroid carcinoma by bioinformatics analysis.New insight into the treatment of advanced differentiated thyroid cancer.BRAF V600E mutation in anaplastic thyroid carcinomas and their accompanying differentiated carcinomas Genetic alterations in thyroid cancer: the role of mouse models.Evolving approaches to patients with advanced differentiated thyroid cancer Molecular genetics of papillary thyroid carcinoma: great expectations.Clinical prognosis in BRAF-mutated PTC.Recent progress of genome study for anaplastic thyroid cancer.Essential genes in thyroid cancers: focus on fascin.New agents in the treatment for malignancies of the salivary and thyroid glands.The PLK1 inhibitor GSK461364A is effective in poorly differentiated and anaplastic thyroid carcinoma cells, independent of the nature of their driver mutations Thyroid tumors: novel insights from proteomic studies.KRAS and BRAF: drug targets and predictive biomarkers.VE1 immunohistochemical detection of the BRAF V600E mutation in thyroid carcinoma: a review of its usefulness and limitations.Normal vs cancer thyroid stem cells: the road to transformation.New Insights in Thyroid Cancer and p53 Family Proteins.Thyrocyte-specific inactivation of p53 and Pten results in anaplastic thyroid carcinomas faithfully recapitulating human tumors Epigallocatechin-3-gallate induces growth inhibition and apoptosis of human anaplastic thyroid carcinoma cells through suppression of EGFR/ERK pathway and cyclin B1/CDK1 complex.

P2860

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P2860

Evidence that one subset of anaplastic thyroid carcinomas are derived from papillary carcinomas due to BRAF and p53 mutations

http://www.wikidata.org/entity/Q28249293

description

2005 nî lūn-bûn

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2005 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Evidence that one subset of an ...... due to BRAF and p53 mutations

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Helen G Ding

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Paolo Gattuso

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Richard A Prinz

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Roderick M Quiros

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Xiulong Xu

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2261-8

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scholarly article

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10.1002/CNCR.21073

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11

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103

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15880523

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