Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
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Long-Term Potentiation and MemoryAllosteric potentiation of quisqualate receptors by a nootropic drug aniracetamIdentification of a Ca2+/calmodulin-dependent protein kinase II regulatory phosphorylation site in non-N-methyl-D-aspartate glutamate receptorsAMPA receptor trafficking and long-term potentiationBi-directional modulation of AMPA receptor unitary conductance by synaptic activityPostsynaptic factors in the expression of long-term potentiation (LTP): increased glutamate receptor binding following LTP induction in vivoModulation of AMPA receptor unitary conductance by synaptic activityStructural basis of long-term potentiation in single dendritic spinesFailure to reverse long-term potentiation by coupling sustained presynaptic activity and N-methyl-D-aspartate receptor blockade.Spatial distribution of potentiated synapses in hippocampus: dependence on cellular mechanisms and network properties.Effects of adrenal cortex hormones on limbic structures: some experimental and clinical correlations related to depression.A biophysical model of bidirectional synaptic plasticity: dependence on AMPA and NMDA receptorsNootropic drugs positively modulate alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid-sensitive glutamate receptors in neuronal cultures.Long-term potentiation of exogenous glutamate responses at single dendritic spines.Synaptic plasticity in rat hippocampal slice cultures: local "Hebbian" conjunction of pre- and postsynaptic stimulation leads to distributed synaptic enhancementPhosphorylation of the alpha-amino-3-hydroxy-5-methylisoxazole4-propionic acid receptor GluR1 subunit by calcium/calmodulin-dependent kinase II.Antidepressant mechanisms: functional and molecular correlates of excitatory amino acid neurotransmission.Expression mechanisms underlying long-term potentiation: a postsynaptic view.Synaptic disinhibition during maintenance of long-term potentiation in the CA1 hippocampal subfield.Developmental synaptic plasticity at the thalamocortical input to barrel cortex: mechanisms and rolesNovel expression mechanism for synaptic potentiation: alignment of presynaptic release site and postsynaptic receptor.Expression of NMDA receptor-dependent LTP in the hippocampus: bridging the divide.Changes in presynaptic function during long-term potentiation.Postsynaptic protein kinase C essential to induction and maintenance of long-term potentiation in the hippocampal CA1 region.NMDA receptor-dependent long-term potentiation comprises a family of temporally overlapping forms of synaptic plasticity that are induced by different patterns of stimulation.Long-term potentiation at single fiber inputs to hippocampal CA1 pyramidal cells.Modulation of DL-alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid/quisqualate receptors by phospholipase A2: a necessary step in long-term potentiation?Review on the role of AMPA receptor nano-organization and dynamic in the properties of synaptic transmission.Discovering long-term potentiation (LTP) - recollections and reflections on what came after.Nitric oxide signaling is recruited as a compensatory mechanism for sustaining synaptic plasticity in Alzheimer's disease mice.Long-term potentiation in the Eocene.High-fat diets induce changes in hippocampal glutamate metabolism and neurotransmission.Quinoxalines interact with the glycine recognition site of NMDA receptors: studies in guinea-pig myenteric plexus and in rat cortical membranes.Inositol hexakisphosphate-mediated regulation of glutamate receptors in rat brain sections.Transient and sustained types of long-term potentiation in the CA1 area of the rat hippocampus.The caveolin scaffolding domain modifies 2-amino-3-hydroxy-5-methyl-4-isoxazole propionate receptor binding properties by inhibiting phospholipase A2 activity.NBQX or topiramate treatment after perinatal hypoxia-induced seizures prevents later increases in seizure-induced neuronal injury.Tetanization-induced pelvic-to-pudendal reflex plasticity in anesthetized rats.Post-ischemic treatment with toki-shakuyaku-san (tang-gui-shao-yao-san) prevents the impairment of spatial memory induced by repeated cerebral ischemia in rats.Focal photolysis of caged glutamate produces long-term depression of hippocampal glutamate receptors.
P2860
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P2860
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
description
1989 nî lūn-bûn
@nan
1989 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@ast
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@en
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@nl
type
label
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@ast
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@en
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@nl
prefLabel
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@ast
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@en
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@nl
P2093
P356
P1433
P1476
Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation
@en
P2093
G L Collingridge
K G Reymann
R A Lester
S N Davies
P2888
P356
10.1038/338500A0
P407
P577
1989-04-06T00:00:00Z
P6179
1043890334