Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice - Wikidata - wikimedia - TriplyDB

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

http://www.wikidata.org/entity/Q28265772

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Oncostatin M promotes bone formation independently of resorption when signaling through leukemia inhibitory factor receptor in mice Oncostatin M receptor-beta mutations underlie familial primary localized cutaneous amyloidosis Helminth infections: the great neglected tropical diseases Biological Treatments in Atopic Dermatitis The involvement of the JAK-STAT signaling pathway in chronic inflammatory skin disease atopic dermatitis Deciphering the complexities of atopic dermatitis: shifting paradigms in treatment approaches STAT6-dependent and -independent mechanisms in Th2 polarization New era of biologic therapeutics in atopic dermatitis Treatment of allergic asthma: modulation of Th2 cells and their responses Chemokines and cytokines network in the pathogenesis of the inflammatory skin diseases: atopic dermatitis, psoriasis and skin mastocytosis Rosacea: The cytokine and chemokine network New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Basic mechanisms of itch.Roles of Mas-related G protein-coupled receptor X2 on mast cell-mediated host defense, pseudoallergic drug reactions, and chronic inflammatory diseases IL-31-IL-31R interactions limit the magnitude of Th2 cytokine-dependent immunity and inflammation following intestinal helminth infection Neural processing of itch.The transcription factor EPAS1 links DOCK8 deficiency to atopic skin inflammation via IL-31 induction Recent insights into atopic dermatitis and implications for management of infectious complications.Associations between Interleukin-31 Gene Polymorphisms and Dilated Cardiomyopathy in a Chinese Population.Mast cells in atopic dermatitis Tlx3 Function in the Dorsal Root Ganglion is Pivotal to Itch and Pain Sensations.Itch mechanisms and circuits.A novel missense mutation in oncostatin M receptor beta causing primary localized cutaneous amyloidosis.25-Hydroxyvitamin D, IL-31, and IL-33 in children with allergic disease of the airways Animal models of atopic dermatitis.A mechanism for the initiation of allergen-induced T helper type 2 responses IL-31 associated with coronary artery lesion formation in Kawasaki disease Retinoid X receptor ablation in adult mouse keratinocytes generates an atopic dermatitis triggered by thymic stromal lymphopoietin Activation of eosinophils interacting with dermal fibroblasts by pruritogenic cytokine IL-31 and alarmin IL-33: implications in atopic dermatitis IL-27 structural analysis demonstrates similarities with ciliary neurotrophic factor (CNTF) and leads to the identification of antagonistic variants Novel IL31RA gene mutation and ancestral OSMR mutant allele in familial primary cutaneous amyloidosis.Inhibition of a novel fibrogenic factor Tl1a reverses established colonic fibrosis.Oclacitinib (APOQUEL(®)) is a novel Janus kinase inhibitor with activity against cytokines involved in allergy.Interleukin-31: a novel diagnostic marker of allergic diseases.Target-oriented therapy: Emerging drugs for atopic dermatitis.Interleukin-31 pathway and its role in atopic dermatitis: a systematic review.Defective IL-10 signaling in hyper-IgE syndrome results in impaired generation of tolerogenic dendritic cells and induced regulatory T cells.Anti-IL-31 receptor antibody is shown to be a potential therapeutic option for treating itch and dermatitis in mice Descending control of itch transmission by the serotonergic system via 5-HT1A-facilitated GRP-GRPR signaling Atopic dermatitis in children: clinical features, pathophysiology, and treatment

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P2860

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

http://www.wikidata.org/entity/Q28265772

description

2004 nî lūn-bûn

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2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2004 թվականի հուլիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@ast

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@en

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@nl

type

label

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@ast

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@en

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@nl

prefLabel

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@ast

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@en

Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@nl

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Nature Immunology

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Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice

@en

P2093

Angela Hammond

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Brandon Harder

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Cindy Sprecher

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Cosette LeCiel

http://www.w3.org/2001/XMLSchema#string

Dennis L Dong

http://www.w3.org/2001/XMLSchema#string

Don Foster

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Francis J Grant

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Harald S Haugen

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Heather Day

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Jane A Gross

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P2860

TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

A novel type I cytokine receptor is expressed on monocytes, signals proliferation, and activates STAT-3 and STAT-5

Toll-like receptor 4 is required for optimal development of Th2 immune responses: role of dendritic cells

A simple and very efficient method for generating cDNA libraries

GPL, a novel cytokine receptor related to GP130 and leukemia inhibitory factor receptor

Inflammatory skin diseases, T cells, and immune surveillance.

Control of B cell development by Ras-mediated activation of Raf.

IL-18 contributes to the spontaneous development of atopic dermatitis-like inflammatory skin lesion independently of IgE/stat6 under specific pathogen-free conditions

Pathophysiology of pruritus in atopic dermatitis: an overview.

Molecular phylogeny within type I cytokines and their cognate receptors.

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752-60

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2243411

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10.1038/NI1084

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7

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5

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2004-07-01T00:00:00Z

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8521978

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15184896

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