Inhibition of GSK-3beta leading to the loss of phosphorylated MAP-1B is an early event in axonal remodelling induced by WNT-7a or lithium
about
WNT7a induces E-cadherin in lung cancer cellsProtein kinase MARK/PAR-1 is required for neurite outgrowth and establishment of neuronal polarity.Evidence that the dephosphorylation of Ser(535) in the epsilon-subunit of eukaryotic initiation factor (eIF) 2B is insufficient for the activation of eIF2B by insulinKLHL1/MRP2 mediates neurite outgrowth in a glycogen synthase kinase 3beta-dependent mannerThe Presynaptic Microtubule Cytoskeleton in Physiological and Pathological Conditions: Lessons from Drosophila Fragile X Syndrome and Hereditary Spastic ParaplegiasMolecular and biochemical trajectories from diabetes to Alzheimer's disease: A critical appraisalWnts in action: from synapse formation to synaptic maintenanceBranch management: mechanisms of axon branching in the developing vertebrate CNSIntraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During DiseaseThe postsynaptic adenomatous polyposis coli (APC) multiprotein complex is required for localizing neuroligin and neurexin to neuronal nicotinic synapses in vivoWnt regulates axon behavior through changes in microtubule growth directionality: a new role for adenomatous polyposis coli.A divergent canonical WNT-signaling pathway regulates microtubule dynamics: dishevelled signals locally to stabilize microtubulesGlycogen synthase kinase-3beta haploinsufficiency mimics the behavioral and molecular effects of lithiumLithium alters the morphology of neurites regenerating from cultured adult spiral ganglion neuronsMicrotubule affinity-regulating kinase 2 functions downstream of the PAR-3/PAR-6/atypical PKC complex in regulating hippocampal neuronal polarityControl of neuronal ion channel function by glycogen synthase kinase-3: new prospective for an old kinase.GSK-3: Functional Insights from Cell Biology and Animal Models.Abnormalities in brain structure and behavior in GSK-3alpha mutant miceElevated glycogen synthase kinase-3 activity in Fragile X mice: key metabolic regulator with evidence for treatment potential.c-Jun N-terminal kinase (JNK) cooperates with Gsk3beta to regulate Dishevelled-mediated microtubule stability.Roles of Wnt proteins in neural development and maintenance.Silencing of Wnt signaling and activation of multiple metabolic pathways in response to thyroid hormone-stimulated cell proliferation.Essential roles for GSK-3s and GSK-3-primed substrates in neurotrophin-induced and hippocampal axon growth.GSK3 takes centre stage more than 20 years after its discovery.Priming-dependent phosphorylation and regulation of the tumor suppressor pVHL by glycogen synthase kinase 3.What Are the bona fide GSK3 Substrates?Wnts and TGF beta in synaptogenesis: old friends signalling at new places.Repulsive axon guidance by Draxin is mediated by protein Kinase B (Akt), glycogen synthase kinase-3β (GSK-3β) and microtubule-associated protein 1B.Glycogen synthase kinase-3: a promising therapeutic target for fragile x syndromeThe in vivo roles of STEF/Tiam1, Rac1 and JNK in cortical neuronal migrationMaternal Wnt/STOP signaling promotes cell division during early Xenopus embryogenesisGlycogen synthase kinase-3 (GSK3) in psychiatric diseases and therapeutic interventions.Wnt transmembrane signaling and long-term spatial memory.Growth cone travel in space and time: the cellular ensemble of cytoskeleton, adhesion, and membrane.Wnt signaling in neuromuscular junction developmentActivating the translational repressor 4E-BP or reducing S6K-GSK3β activity prevents accelerated axon growth induced by hyperactive mTOR in vivo.Temporal activation of β-catenin signaling in the chondrogenic process of mesenchymal stem cells affects the phenotype of the cartilage generated.MAP1B is required for axon guidance and Is involved in the development of the central and peripheral nervous system.Phosphoproteomic differences in major depressive disorder postmortem brains indicate effects on synaptic function.
P2860
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P2860
Inhibition of GSK-3beta leading to the loss of phosphorylated MAP-1B is an early event in axonal remodelling induced by WNT-7a or lithium
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@ast
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@en
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@nl
type
label
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@ast
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@en
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@nl
prefLabel
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@ast
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@en
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@nl
P2093
P3181
P1476
Inhibition of GSK-3beta leadin ...... g induced by WNT-7a or lithium
@en
P2093
P C Salinas
P R Gordon-Weeks
P304
P3181
P407
P478
111 ( Pt 10)
P577
1998-05-01T00:00:00Z