Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
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The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and AgingIncreased 4E-BP1 Expression Protects against Diet-Induced Obesity and Insulin Resistance in Male Mice.Moderate lifelong overexpression of tuberous sclerosis complex 1 (TSC1) improves health and survival in miceAging and Autophagy in the HeartRapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island.Rapamycin increases grip strength and attenuates age-related decline in maximal running distance in old low capacity runner rats.Comparative idiosyncrasies in life extension by reduced mTOR signalling and its distinctiveness from dietary restrictionAge related increase in mTOR activity contributes to the pathological changes in ovarian surface epithelium.Voluntary resistance wheel exercise from mid-life prevents sarcopenia and increases markers of mitochondrial function and autophagy in muscles of old male and female C57BL/6J mice.The underlying mechanism of proinflammatory NF-κB activation by the mTORC2/Akt/IKKα pathway during skin aging.Sexual dimorphism in glioma glycolysis underlies sex differences in survivalSex differences in aging: genomic instability.mTOR drives cerebral blood flow and memory deficits in LDLR(-/-) mice modeling atherosclerosis and vascular cognitive impairment.Alcoholic Cardiomyopathy: Disrupted Protein Balance and Impaired Cardiomyocyte Contractility.Life history evolution, reproduction, and the origins of sex-dependent aging and longevity.Evidence that a mitochondrial death spiral underlies antagonistic pleiotropy.Simulation of Cellular Energy Restriction in Quiescence (ERiQ)-A Theoretical Model for Aging.PTEN deletion increases hippocampal granule cell excitability in male and female mice.Restoration of metabolic health by decreased consumption of branched-chain amino acids.Deletion of Nrip1 extends female mice longevity, increases autophagy, and delays cell senescence.Evolution of sex-specific pace-of-life syndromes: genetic architecture and physiological mechanisms.Short-term methionine deprivation improves metabolic health via sexually dimorphic, mTORC1-independent mechanisms.Loss of Malat1 does not modify age- or diet-induced adipose tissue accretion and insulin resistance in mice.
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P2860
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
description
2016 nî lūn-bûn
@nan
2016 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@ast
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@en
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@nl
type
label
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@ast
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@en
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@nl
prefLabel
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@ast
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@en
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@nl
P2093
P2860
P3181
P356
P1433
P1476
Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice
@en
P2093
Emma L Baar
Irene M Ong
Kathryn A Carbajal
P2860
P304
P3181
P356
10.1111/ACEL.12425
P407
P577
2015-11-24T00:00:00Z