GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity - Wikidata - wikimedia - TriplyDB

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

http://www.wikidata.org/entity/Q28275543

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Human Carboxylesterase 2 Reverses Obesity-Induced Diacylglycerol Accumulation and Glucose Intolerance.DGAT1-Dependent Lipid Droplet Biogenesis Protects Mitochondrial Function during Starvation-Induced Autophagy.Activity-based protein profiling for mapping and pharmacologically interrogating proteome-wide ligandable hotspots.Impact of the Tumor Microenvironment on Tumor-Infiltrating Lymphocytes: Focus on Breast Cancer.Prognostic significance of GSTP1 in patients with triple negative breast cancer.Pathway-based discovery of genetic interactions in breast cancer The Ubiquitin Binding Protein TAX1BP1 Mediates Autophagasome Induction and the Metabolic Transition of Activated T Cells.Covalent Ligand Discovery against Druggable Hotspots Targeted by Anti-cancer Natural Products.GFP labeling kinetics of triple-negative human breast cancer by a killer-reporter adenovirus in 3D Gelfoam® histoculture.Metabolic reprogramming ensures cancer cell survival despite oncogenic signaling blockade.High-metastatic triple-negative breast-cancer variants selected in vivo become chemoresistant in vitro.Lysosomal cholesterol activates mTORC1 via an SLC38A9-Niemann-Pick C1 signaling complex.Paternal chromosome loss and metabolic crisis contribute to hybrid inviability in Xenopus.Metabolic advantages and vulnerabilities in brain metastases.miR-133b reverses cisplatin resistance by targeting GSTP1 in cisplatin-resistant lung cancer cells.Discovering Targets of Non-enzymatic Acylation by Thioester Reactivity Profiling.Identification of methylation sites and signature genes with prognostic value for luminal breast cancer.GSTM3 and GSTP1: novel players driving tumor progression in cervical cancer.Transmembrane TNF-alpha promotes chemoresistance in breast cancer cells.Metabolomic Markers of Phthalate Exposure in Plasma and Urine of Pregnant Women Stable Isotope-Resolved Metabolomic Differences between Hormone-Responsive and Triple-Negative Breast Cancer Cell Lines

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P2860

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

http://www.wikidata.org/entity/Q28275543

description

2016 nî lūn-bûn

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2016 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2016 թվականի մայիսին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@ast

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@en

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

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type

label

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

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GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@en

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@nl

prefLabel

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@ast

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

@en

GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

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J.CHEMBIOL.2016.03.017

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Cell chemical biology

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GSTP1 Is a Driver of Triple-Negative Breast Cancer Cell Metabolism and Pathogenicity

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P2093

Andrei Goga

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Daniel K Nomura

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David K Miyamoto

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Elizabeth A Grossman

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Eranthie Weerapana

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Lucky Ding

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Roman Camarda

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Sharon M Louie

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P2860

Metabolomics annotates ABHD3 as a physiologic regulator of medium-chain phospholipids

Transitions between epithelial and mesenchymal states: acquisition of malignant and stem cell traits

Interactive XCMS Online: simplifying advanced metabolomic data processing and subsequent statistical analyses

Triple-negative breast cancer in African-American women: disparities versus biology.

Inositol phosphate recycling regulates glycolytic and lipid metabolism that drives cancer aggressiveness.

Monoacylglycerol lipase regulates a fatty acid network that promotes cancer pathogenesis.

Quantitative determinants of aerobic glycolysis identify flux through the enzyme GAPDH as a limiting step.

Metabolic profiling reveals PAFAH1B3 as a critical driver of breast cancer pathogenicity.

Glutathione transferases as mediators of signaling pathways involved in cell proliferation and cell death.

The role of glutathione-S-transferase in anti-cancer drug resistance.

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567-78

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10.1016/J.CHEMBIOL.2016.03.017

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5

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Tucker R Huffman

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2016-05-19T00:00:00Z

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27185638

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4876719

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