Identification of GPR99 protein as a potential third cysteinyl leukotriene receptor with a preference for leukotriene E4 ligand - Wikidata - wikimedia - TriplyDB

Identification of GPR99 protein as a potential third cysteinyl leukotriene receptor with a preference for leukotriene E4 ligand

Identification of GPR99 protein as a potential third cysteinyl leukotriene receptor with a preference for leukotriene E4 ligand

http://www.wikidata.org/entity/Q28287399

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Aspirin-exacerbated respiratory disease: pathophysiological insights and clinical advances Lipid mediators and allergic diseases Cysteinyl Leukotrienes Pathway Genes, Atopic Asthma and Drug Response: From Population Isolates to Large Genome-Wide Association Studies Evaluation of Cysteinyl Leukotriene Signaling as a Therapeutic Target for Colorectal Cancer Biological effects of leukotriene E4 on eosinophils Cysteinyl leukotrienes and their receptors; emerging concepts Leukotriene E4 elicits respiratory epithelial cell mucin release through the G-protein-coupled receptor, GPR99 Lipid Mediators of Allergic Disease: Pathways, Treatments, and Emerging Therapeutic Targets Cysteinyl Leukotrienes and Their Receptors: Emerging Therapeutic Targets in Central Nervous System Disorders A review on leukotrienes and their receptors with reference to asthma The eosinophil chemoattractant 5-oxo-ETE and the OXE receptor Update on leukotriene, lipoxin and oxoeicosanoid receptors: IUPHAR Review 7 Eosinophils and Mast Cells in Aspirin-Exacerbated Respiratory Disease.Factors driving the aspirin exacerbated respiratory disease phenotype.Leukotriene E4 is a full functional agonist for human cysteinyl leukotriene type 1 receptor-dependent gene expression.Prostaglandin E2 resistance in granulocytes from patients with aspirin-exacerbated respiratory disease.Platelet-driven leukotriene C4-mediated airway inflammation in mice is aspirin-sensitive and depends on T prostanoid receptors.Prostaglandin D₂: a dominant mediator of aspirin-exacerbated respiratory disease.Prostaglandin D2 and leukotriene E4 synergize to stimulate diverse TH2 functions and TH2 cell/neutrophil crosstalk.Phenotype-based Discovery of 2-[(E)-2-(Quinolin-2-yl)vinyl]phenol as a Novel Regulator of Ocular Angiogenesis.Endogenous secreted phospholipase A2 group X regulates cysteinyl leukotrienes synthesis by human eosinophils.P2Y12 antagonist attenuates eosinophilic inflammation and airway hyperresponsiveness in a mouse model of asthma.Asthma genetics and personalised medicine.Prostaglandin E2 deficiency causes a phenotype of aspirin sensitivity that depends on platelets and cysteinyl leukotrienes.Leukotriene B4 receptor type 2 protects against pneumolysin-dependent acute lung injury.Leukotrienes provide an NFAT-dependent signal that synergizes with IL-33 to activate ILC2s.Pharmacological Properties and Biological Functions of the GPR17 Receptor, a Potential Target for Neuro-Regenerative Medicine.Molecular mechanisms of target recognition by lipid GPCRs: relevance for cancer.Leukotriene C4 Potentiates IL-33-Induced Group 2 Innate Lymphoid Cell Activation and Lung Inflammation.A Quininib Analogue and Cysteinyl Leukotriene Receptor Antagonist Inhibits Vascular Endothelial Growth Factor (VEGF)-independent Angiogenesis and Exerts an Additive Antiangiogenic Response with Bevacizumab.The oxoglutarate receptor 1 (OXGR1) modulates pressure overload-induced cardiac hypertrophy in mice.Leukotriene D4 and prostaglandin E2 signals synergize and potentiate vascular inflammation in a mast cell-dependent manner through cysteinyl leukotriene receptor 1 and E-prostanoid receptor 3.Development of novel aminothiazole-comprising 5-LO inhibitors.Enhanced cysteinyl-leukotriene type 1 receptor expression in T cells from house dust mite-allergic individuals following stimulation with Der p.Phosphorylation of Leukotriene C4 Synthase at Serine 36 Impairs Catalytic Activity.Commentary: Mutual interaction of basophils and T cells in chronic inflammatory diseases.Cysteinyl Leukotrienes in Eosinophil Biology: Functional Roles and Therapeutic Perspectives in Eosinophilic Disorders Role of the Cysteinyl Leukotrienes in the Pathogenesis and Progression of Cardiovascular Diseases.Cysteinyl leukotriene E4 activates human group 2 innate lymphoid cells and enhances the effect of prostaglandin D2 and epithelial cytokines.Cutting edge: Leukotriene C4 activates mouse platelets in plasma exclusively through the type 2 cysteinyl leukotriene receptor.

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Identification of GPR99 protein as a potential third cysteinyl leukotriene receptor with a preference for leukotriene E4 ligand

http://www.wikidata.org/entity/Q28287399

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2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013年の論文

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Identification of GPR99 protei ...... ence for leukotriene E4 ligand

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Akiko Maekawa

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K Frank Austen

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Yoshihide Kanaoka

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P2860

GPR99, a new G protein-coupled receptor with homology to a new subgroup of nucleotide receptors

Characterization of the human cysteinyl leukotriene CysLT1 receptor

Functional recognition of a distinct receptor preferential for leukotriene E4 in mice lacking the cysteinyl leukotriene 1 and 2 receptors

Improvement of aspirin-intolerant asthma by montelukast, a leukotriene antagonist: a randomized, double-blind, placebo-controlled trial

Expression and localization of the cysteinyl leukotriene 1 receptor in human nasal mucosa

Leukotriene E4 elimination and metabolism in normal human subjects

Identification and characterization of a cell-surface receptor, P2Y15, for AMP and adenosine

The leukotriene E4 puzzle: finding the missing pieces and revealing the pathobiologic implications

Citric acid cycle intermediates as ligands for orphan G-protein-coupled receptors

Characterization of within-subject responses to fluticasone and montelukast in childhood asthma

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