Isolation, characterization, and organ-specific expression of two novel human zinc finger genes related to the Drosophila gene spalt - Wikidata - wikimedia - TriplyDB

Isolation, characterization, and organ-specific expression of two novel human zinc finger genes related to the Drosophila gene spalt

Isolation, characterization, and organ-specific expression of two novel human zinc finger genes related to the Drosophila gene spalt

http://www.wikidata.org/entity/Q28299972

about

Sall1 regulates mitral cell development and olfactory nerve extension in the developing olfactory bulb Mutation of SALL2 causes recessive ocular coloboma in humans and mice Transcriptional activation of the SALL1 by the human SIX1 homeodomain during kidney development Molecular analysis of SALL1 mutations in Townes-Brocks syndrome.p150(Sal2) is a p53-independent regulator of p21(WAF1/CIP)Duane radial ray syndrome (Okihiro syndrome) maps to 20q13 and results from mutations in SALL4, a new member of the SAL family Bmi-1 is a target gene for SALL4 in hematopoietic and leukemic cells Mutations at the SALL4 locus on chromosome 20 result in a range of clinically overlapping phenotypes, including Okihiro syndrome, Holt-Oram syndrome, acro-renal-ocular syndrome, and patients previously reported to represent thalidomide embryopathy Tissue microarray validation of epidermal growth factor receptor and SALL2 in synovial sarcoma with comparison to tumors of similar histology SALL4, a novel oncogene, is constitutively expressed in human acute myeloid leukemia (AML) and induces AML in transgenic mice The conserved glutamine-rich region of chick csal1 and csal3 mediates protein interactions with other spalt family members. Implications for Townes-Brocks syndrome Stem cell factor SALL4 represses the transcriptions of PTEN and SALL1 through an epigenetic repressor complex The expression of SALL4 in patients with gliomas: high level of SALL4 expression is correlated with poor outcome SALL4, the missing link between stem cells, development and cancer SALL4 is a key regulator of survival and apoptosis in human leukemic cells Targeting transcription factor SALL4 in acute myeloid leukemia by interrupting its interaction with an epigenetic complex A Sall4 mutant mouse model useful for studying the role of Sall4 in early embryonic development and organogenesis Role of SALL4 in the progression and metastasis of colorectal cancer Histone lysine-specific demethylase 1 (LSD1) protein is involved in Sal-like protein 4 (SALL4)-mediated transcriptional repression in hematopoietic stem cells Mouse homolog of SALL1, a causative gene for Townes-Brocks syndrome, binds to A/T-rich sequences in pericentric heterochromatin via its C-terminal zinc finger domains Sall3 is required for the terminal maturation of olfactory glomerular interneurons Zinc finger protein sall2 is not essential for embryonic and kidney development Ex-vivo expansion of nonhuman primate CD34(+) cells by stem cell factor Sall4B Drosophila spalt/spalt-related mutants exhibit Townes-Brocks' syndrome phenotypes.Townes-Brocks syndrome.Oncofetal gene SALL4 and prognosis in cancer: A systematic review with meta-analysis.A tumor host range selection procedure identifies p150(sal2) as a target of polyoma virus large T antigen.Fluorescence- and magnetic-activated cell sorting strategies to isolate and enrich human spermatogonial stem cells.SALL1 mutations in Townes-Brocks syndrome and related disorders.Townes-Brocks syndrome versus expanded spectrum hemifacial microsomia: review of eight patients and further evidence of a "hot spot" for mutation in the SALL1 gene.Expression of transcript factors SALL4 and OCT4 in a subset of non-small cell lung carcinomas (NSCLC).SALL4 expression in gonocytes and spermatogonial clones of postnatal mouse testes Transcriptional and post-translational regulation of the quiescence factor and putative tumor suppressor p150(Sal2).Enhanced self-renewal of hematopoietic stem/progenitor cells mediated by the stem cell gene Sall4 Knockdown of SALL4 Protein Enhances All-trans Retinoic Acid-induced Cellular Differentiation in Acute Myeloid Leukemia Cells The role of HSAL (SALL) genes in proliferation and differentiation in normal hematopoiesis and leukemogenesis.Stem cell gene SALL4 suppresses transcription through recruitment of DNA methyltransferases.Loss of the Sall3 gene leads to palate deficiency, abnormalities in cranial nerves, and perinatal lethality.Diversified clinical presentations associated with a novel sal-like 4 gene mutation in a Chinese pedigree with Duane retraction syndrome.Genome-wide analysis reveals Sall4 to be a major regulator of pluripotency in murine-embryonic stem cells.

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P2860

Isolation, characterization, and organ-specific expression of two novel human zinc finger genes related to the Drosophila gene spalt

http://www.wikidata.org/entity/Q28299972

description

1996 nî lūn-bûn

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1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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1996年の論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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