Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse - Wikidata - wikimedia - TriplyDB

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

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Whole-genome profiling of mutagenesis in Caenorhabditis elegans A focused and efficient genetic screening strategy in the mouse: identification of mutations that disrupt cortical development Efficient gene-driven germ-line point mutagenesis of C57BL/6J mice Cilia/Ift protein and motor -related bone diseases and mouse models Spontaneous 8bp Deletion in Nbeal2 Recapitulates the Gray Platelet Syndrome in Mice Unlocking the bottleneck in forward genetics using whole-genome sequencing and identity by descent to isolate causative mutations Transgenic and gene knockout mice in gastric cancer research The construction of transgenic and gene knockout/knockin mouse models of human disease Mutagenesis and mapping of a mouse gene, Clock, essential for circadian behavior An Oak Ridge legacy: the specific locus test and its role in mouse mutagenesis Pahhph-5: a mouse mutant deficient in phenylalanine hydroxylase Point mutations in the murine fumarylacetoacetate hydrolase gene: Animal models for the human genetic disorder hereditary tyrosinemia type 1 Mice with genetic gamma-glutamyl transpeptidase deficiency exhibit glutathionuria, severe growth failure, reduced life spans, and infertility A missense mutation in the Capza3 gene and disruption of F-actin organization in spermatids of repro32 infertile male mice Analysis of mouse embryonic patterning and morphogenesis by forward genetics Mammalian circadian biology: elucidating genome-wide levels of temporal organization Nonrandom distribution of structural mutants in ethylnitrosourea-treated cultured human lymphoblastoid cells Male hypogonadism and germ cell loss caused by a mutation in Polo-like kinase 4.A forward genetic screen with a thalamocortical axon reporter mouse yields novel neurodevelopment mutants and a distinct emx2 mutant phenotype.Phenotype screening for genetically determined age-onset disorders and increased longevity in ENU-mutagenized mice Mouse homologues of human hereditary disease.An allelic series of mutations in Smad2 and Smad4 identified in a genotype-based screen of N-ethyl-N- nitrosourea-mutagenized mouse embryonic stem cells.The expanding role of mouse genetics for understanding human biology and disease Novel ENU-induced point mutation in scavenger receptor class B, member 1, results in liver specific loss of SCARB1 protein.Development of an enhanced GFP-based dual-color reporter to facilitate genetic screens for the recovery of mutations in mice Mapping to molecular resolution in the T to H-2 region of the mouse genome with a nested set of meiotic recombinants.Recovery of induced mutations for X chromosome-linked muscular dystrophy in mice.Isolated spermatozoa as indicators of mutations transmitted to progeny Pilot study of large-scale production of mutant pigs by ENU mutagenesis Functional annotation of mammalian genomic DNA sequence by chemical mutagenesis: a fine-structure genetic mutation map of a 1- to 2-cM segment of mouse chromosome 7 corresponding to human chromosome 11p14-p15.Modifier screens in the mouse: time to move forward with reverse genetics.N-ethyl-N-nitrosourea mutagenesis: boarding the mouse mutant express.Ethylnitrosourea mutagenesis and the isolation of mutant alleles for specific genes located in the T region of mouse chromosome 17.A mouse beta-globin mutant that is an exact model of hemoglobin Rainier in man.Molecular genetic analysis of the dilute-short ear (d-se) region of the mouse.Molecular genetics of Mus musculus: point mutagenesis and millimorgans.Detection of new MHC mutations in mice by skin grafting, tumor transplantation and monoclonal antibodies: a comparison hph-1: a mouse mutant with hereditary hyperphenylalaninemia induced by ethylnitrosourea mutagenesis Genetic analysis of mouse t haplotypes using mutations induced by ethylnitrosourea mutagenesis: the order of T and qk is inverted in t mutants.Mouse models of human phenylketonuria.

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Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

http://www.wikidata.org/entity/Q28323835

description

1979 nî lūn-bûn

@nan

1979 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

1979 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

1979年の論文

@ja

1979年論文

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1979年論文

@zh-hant

1979年論文

@zh-hk

1979年論文

@zh-mo

1979年論文

@zh-tw

1979年论文

@wuu

name

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@ast

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@en

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@nl

type

label

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@ast

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@en

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@nl

prefLabel

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@ast

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@en

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@nl

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PNAS.76.11.5818

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse

@en

P2093

E L Phipps

http://www.w3.org/2001/XMLSchema#string

E M Kelly

http://www.w3.org/2001/XMLSchema#string

J W Bangham

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P R Hunsicker

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S C Maddux

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W L Russell

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Dominant mutations affecting the skeleton in offspring of x-irradiated male mice.

Mutagenicity of nitrosamides and nitrosamidines in micro-organisms and plants.

Isolation and identification of products from alkylation of nucleic acids: ethyl- and isopropyl-purines.

Induced skeletal mutations.

Procarbazine-induced specific-locus mutations in male mice.

Concentration-effect studies with MMS, TEB, 2, 4, 6-TriCl-PDMT, and DEN on the induction of dominant and recessive lethals, chromosome loss and translocations in Drosophila sterm.

The relation between reaction kinetics and mutagenic action of mono-functional alkylating agents in higher eukaryotic systems. I. Recessive lethal mutations and translocations in Drosophila.

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