Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
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Cannabidiol, extracted from Cannabis sativa, selectively inhibits inflammatory hypermotility in miceAdvances in the discovery of N-acylethanolamine acid amidase inhibitorsInitial insight into the function of the lysosomal 66.3 kDa protein from mouse by means of X-ray crystallographyPalmitoylethanolamide for the treatment of pain: pharmacokinetics, safety and efficacyInhibitory effect of cannabichromene, a major non-psychotropic cannabinoid extracted from Cannabis sativa, on inflammation-induced hypermotility in micePalmitoylethanolamide counteracts reactive astrogliosis induced by β-amyloid peptideThe pharmacology of palmitoylethanolamide and first data on the therapeutic efficacy of some of its new formulations.Administration of URB597, oleoylethanolamide or palmitoylethanolamide increases waking and dopamine in rats.The association of N-palmitoylethanolamine with the FAAH inhibitor URB597 impairs melanoma growth through a supra-additive action.Palmitoylethanolamide regulates development of intestinal radiation injury in a mast cell-dependent mannerPalmitoylethanolamide normalizes intestinal motility in a model of post-inflammatory accelerated transit: involvement of CB₁ receptors and TRPV1 channels.The hallucinogenic herb Salvia divinorum and its active ingredient salvinorin A reduce inflammation-induced hypermotility in mice.Proinflammatory stimuli control N-acylphosphatidylethanolamine-specific phospholipase D expression in macrophages.Palmitoylethanolamide, a naturally occurring lipid, is an orally effective intestinal anti-inflammatory agent.Cellular viability effects of fatty acid amide hydrolase inhibition on cerebellar neuronsInvolvement of cannabinoid receptors in gut motility and visceral perception.Neural contractions in colonic strips from patients with diverticular disease: role of endocannabinoids and substance PInhibiting fatty acid amide hydrolase normalizes endotoxin-induced enhanced gastrointestinal motility in mice.Palmitoylethanolamide Modulates Inflammation-Associated Vascular Endothelial Growth Factor (VEGF) Signaling via the Akt/mTOR Pathway in a Selective Peroxisome Proliferator-Activated Receptor Alpha (PPAR-α)-Dependent Manner.N-Palmitoylethanolamine Prevents the Run-down of Amplitudes in Cortical Spreading Depression Possibly Implicating Proinflammatory Cytokine Release.Inhibitory effect of salvinorin A, from Salvia divinorum, on ileitis-induced hypermotility: cross-talk between kappa-opioid and cannabinoid CB(1) receptors.Endocannabinoid and cannabinoid-like fatty acid amide levels correlate with pain-related symptoms in patients with IBS-D and IBS-C: a pilot study.Selective N-acylethanolamine-hydrolyzing acid amidase inhibition reveals a key role for endogenous palmitoylethanolamide in inflammation.The cannabinoid receptor 1 and its role in influencing peripheral metabolism.Role of G protein-coupled orphan receptors in intestinal inflammation: novel targets in inflammatory bowel diseases.N-Acylethanolamine-hydrolyzing acid amidase inhibition increases colon N-palmitoylethanolamine levels and counteracts murine colitis.Macrophage-derived lipid agonists of PPAR-α as intrinsic controllers of inflammation.Cannabinoid Receptors in Regulating the GI Tract: Experimental Evidence and Therapeutic Relevance.The gastrointestinal tract - a central organ of cannabinoid signaling in health and disease.Endocannabinoid-related lipids are increased during an episode of cyclic vomiting syndrome.Cannabinoid Receptor 2 Modulates Susceptibility to Experimental Cerebral Malaria through a CCL17-dependent Mechanism.Effect of Boswellia serrata on intestinal motility in rodents: inhibition of diarrhoea without constipation.Cannabinoid Modulation of Memory Consolidation in Rats: Beyond the Role of Cannabinoid Receptor Subtype 1Involvement of cannabinoid receptors in inflammatory hypersensitivity to colonic distension in rats.An open-label pilot study of cannabis-based extracts for bladder dysfunction in advanced multiple sclerosis.Endocannabinoid overactivity and intestinal inflammation.Investigation on the relationship between cannabinoid CB1 and opioid receptors in gastrointestinal motility in mice.Effect on cancer cell proliferation of palmitoylethanolamide, a fatty acid amide interacting with both the cannabinoid and vanilloid signalling systems.An Important Role for N-Acylethanolamine Acid Amidase in the Complete Freund's Adjuvant Rat Model of Arthritis.Palmitoylethanolamide Supplementation during Sensitization Prevents Airway Allergic Symptoms in the Mouse.
P2860
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P2860
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
description
2001 nî lūn-bûn
@nan
2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
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2001年论文
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name
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@ast
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@en
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@nl
type
label
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@ast
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@en
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@nl
prefLabel
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@ast
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@en
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@nl
P2093
P2860
P3181
P356
P1476
Inhibitory effect of palmitoylethanolamide on gastrointestinal motility in mice
@en
P2093
V Di Marzo
P2860
P304
P3181
P356
10.1038/SJ.BJP.0704339
P407
P577
2001-11-01T00:00:00Z