Differential cardiovascular responses to blockade of nNOS or iNOS in rostral ventrolateral medulla of the rat - Wikidata - wikimedia - TriplyDB

Differential cardiovascular responses to blockade of nNOS or iNOS in rostral ventrolateral medulla of the rat

Differential cardiovascular responses to blockade of nNOS or iNOS in rostral ventrolateral medulla of the rat

http://www.wikidata.org/entity/Q28366601

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Brain stem adenosine receptors modulate centrally mediated hypotensive responses in conscious rats: A review Brain-derived neurotrophic factor ameliorates brain stem cardiovascular dysregulation during experimental temporal lobe status epilepticus Heat shock protein 60 or 70 activates nitric-oxide synthase (NOS) I- and inhibits NOS II-associated signaling and depresses the mitochondrial apoptotic cascade during brain stem death A review of flux considerations for in vivo neurochemical measurements Neuronal nitric oxide synthase-dependent elevation in adiponectin in the rostral ventrolateral medulla underlies g protein-coupled receptor 18-mediated hypotension in conscious rats.Differential engagements of glutamate and GABA receptors in cardiovascular actions of endogenous nNOS or iNOS at rostral ventrolateral medulla of rats.Differential contributions of nitric oxide synthase isoforms at hippocampal formation to negative feedback regulation of penile erection in the rat Differential involvement of various sources of reactive oxygen species in thyroxin-induced hemodynamic changes and contractile dysfunction of the heart and diaphragm muscles.Programming blood pressure in adult SHR by shifting perinatal balance of NO and reactive oxygen species toward NO: the inverted Barker phenomenon.Nontranscriptional activation of PI3K/Akt signaling mediates hypotensive effect following activation of estrogen receptor β in the rostral ventrolateral medulla of rats.Neuroinflammation and oxidative stress in rostral ventrolateral medulla contribute to neurogenic hypertension induced by systemic inflammation Redox-sensitive oxidation and phosphorylation of PTEN contribute to enhanced activation of PI3K/Akt signaling in rostral ventrolateral medulla and neurogenic hypertension in spontaneously hypertensive rats The insulin signaling cascade from nematodes to mammals: insights into innate immunity of Anopheles mosquitoes to malaria parasite infection.Brain superoxide as a key regulator of the cardiovascular response to emotional stress in rabbits.Swimming Training Modulates Nitric Oxide-Glutamate Interaction in the Rostral Ventrolateral Medulla in Normotensive Conscious Rats Activation of estrogen receptor beta-dependent nitric oxide signaling mediates the hypotensive effects of estrogen in the rostral ventrolateral medulla of anesthetized rats Nitric oxide in rostral ventrolateral medulla regulates cardiac-sympathetic reflexes: role of synthase isoforms.Regulation of the sympathetic nervous system by nitric oxide and oxidative stress in the rostral ventrolateral medulla: 2012 Academic Conference Award from the Japanese Society of Hypertension.Implicating the potential role of orexin in hypertension.Exercise training restores oxidative stress and nitric oxide synthases in the rostral ventrolateral medulla of renovascular hypertensive rats.Age-dependent redox status in the brain stem of NO-deficient hypertensive rats.Endogenous nitric oxide derived from NOS I or II in thoracic spinal cord exerts opposing tonic modulation on sympathetic vasomotor tone via disparate mechanisms in anesthetized rats.Astrocytic iNOS-dependent enhancement of synaptic release in mouse neocortex.Sympathoinhibitory mechanism of moxonidine: role of the inducible nitric oxide synthase in the rostral ventrolateral medulla.Role of inducible nitric oxide synthase in rostral ventrolateral medulla in blood pressure regulation in spontaneously hypertensive rats.Transient middle cerebral artery occlusion and reperfusion alters inducible NOS expression within the ventrolateral medulla and modulates cardiovascular function during static exercise.Hindlimb unloading alters nitric oxide and autonomic control of resting arterial pressure in conscious rats.Sildenafil acts on the central nervous system increasing sympathetic activity.Role of neuronal nitric oxide synthase (nNOS) at medulla in tachycardia induced by repeated administration of ethanol in conscious rats.Modulation of inducible nitric oxide synthase (iNOS) expression and cardiovascular responses during static exercise following iNOS antagonism within the ventrolateral medulla.Spontaneously hypertensive rats have more orexin neurons in the hypothalamus and enhanced orexinergic input and orexin 2 receptor-associated nitric oxide signalling in the rostral ventrolateral medulla.Transcriptional up-regulation of nitric oxide synthase II by nuclear factor-kappaB at rostral ventrolateral medulla in a rat mevinphos intoxication model of brain stem death.Effects of chronic treatment with 7-nitroindazole in hyperthyroid rats.

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P2860

Differential cardiovascular responses to blockade of nNOS or iNOS in rostral ventrolateral medulla of the rat

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2001 nî lūn-bûn

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2001 թուականի Յունիսին հրատարակուած գիտական յօդուած

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P2860

Isolation of nitric oxide synthetase, a calmodulin-requiring enzyme

Characterization of the novel nitric oxide synthase inhibitor 7-nitro indazole and related indazoles: antinociceptive and cardiovascular effects

Role of endothelium-derived nitric oxide in the regulation of blood pressure

Nitric oxide as a secretory product of mammalian cells

Differential roles of iNOS and nNOS at rostral ventrolateral medulla during experimental endotoxemia in the rat.

Role of nitric oxide in the neural control of cardiovascular function.

Inducible nitric oxide synthase gene expression in the brain during systemic inflammation.

Discrimination by the NO-trapping agent, carboxy-PTIO, between NO and the nitrergic transmitter but not between NO and EDRF.

Cytokine-induced synthesis of nitrogen oxides in macrophages: a protective host response to Leishmania and other intracellular pathogens.

Synthesis of nitric oxide in CNS glial cells.

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Aparato circulatorio

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