Suppression subtractive hybridization identifies high glucose levels as a stimulus for expression of connective tissue growth factor and other genes in human mesangial cells
about
Fibrosis in diabetes complications: pathogenic mechanisms and circulating and urinary markersPotential Renoprotective Agents through Inhibiting CTGF/CCN2 in Diabetic NephropathyIdentification of Naf1/ABIN-1 among TNF-alpha-induced expressed genes in human synoviocytes using oligonucleotide microarraysTaking aim at the extracellular matrix: CCN proteins as emerging therapeutic targetsRho-dependent inhibition of the induction of connective tissue growth factor (CTGF) by HMG CoA reductase inhibitors (statins)Role of connective tissue growth factor in the pathogenesis of diabetic nephropathyRegulation of connective tissue growth factor (ccn2; ctgf) gene expression in human mesangial cells: modulation by HMG CoA reductase inhibitors (statins).Urocortin ameliorates diabetic nephropathy in obese db/db miceEffect of high glucose on gene expression in mesangial cells: upregulation of the thiol pathway is an adaptational responseNew susceptibility loci associated with kidney disease in type 1 diabetes.Discovery of genes related to diabetic nephropathy in various animal models by current techniques.Renal (pro)renin receptor contributes to development of diabetic kidney disease through transforming growth factor-β1-connective tissue growth factor signalling cascade.TGFβ and CCN2/CTGF mediate actin related gene expression by differential E2F1/CREB activation.Glomerular expression of connective tissue growth factor mRNA in various renal diseases.Use of serial analysis of gene expression to generate kidney expression libraries.In vivo delivery of Gremlin siRNA plasmid reveals therapeutic potential against diabetic nephropathy by recovering bone morphogenetic protein-7.A GREM1 gene variant associates with diabetic nephropathy.Connective tissue growth factor: just another factor in renal fibrosis?Tubular overexpression of gremlin induces renal damage susceptibility in mice.High glucose-altered gene expression in mesangial cells. Actin-regulatory protein gene expression is triggered by oxidative stress and cytoskeletal disassembly.Protein kinase C and the development of diabetic vascular complications.Gremlin induces cell proliferation and extra cellular matrix accumulation in mouse mesangial cells exposed to high glucose via the ERK1/2 pathwayLower urinary connective tissue growth factor levels and incident CKD stage 3 in the general populationConnective tissue growth factor: an attractive therapeutic target in fibrotic renal disease.Advanced glycation end-products induce connective tissue growth factor-mediated renal fibrosis predominantly through transforming growth factor beta-independent pathway.CCN-2 is up-regulated by and mediates effects of matrix bound advanced glycated end-products in human renal mesangial cells.IHG-1 promotes mitochondrial biogenesis by stabilizing PGC-1α.Gremlin - a putative pathogenic player in progressive renal disease.A tool for biomarker discovery in the urinary proteome: a manually curated human and animal urine protein biomarker database.Glucose stimulation of transforming growth factor-beta bioactivity in mesangial cells is mediated by thrombospondin-1.Bradykinin B1 and B2 receptors both have protective roles in renal ischemia/reperfusion injury.Angiotensin II induces connective tissue growth factor gene expression via calcineurin-dependent pathways.Higher plasma thrombospondin-1 levels in patients with coronary artery disease and diabetes mellitus.Angiotensin II increases connective tissue growth factor in the kidney.Altered Mitochondrial Function, Mitochondrial DNA and Reduced Metabolic Flexibility in Patients With Diabetic Nephropathy.Low-density lipoprotein induced expression of connective tissue growth factor via transactivation of sphingosine 1-phosphate receptors in mesangial cells.The role of connective tissue growth factor (CTGF/CCN2) in skeletogenesis.Decorin deficiency enhances progressive nephropathy in diabetic mice.Bad and good growth factors in the peritoneal cavity.Connective tissue growth factor gene expression and decline in renal function in lupus nephritis
P2860
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P2860
Suppression subtractive hybridization identifies high glucose levels as a stimulus for expression of connective tissue growth factor and other genes in human mesangial cells
description
1999 nî lūn-bûn
@nan
1999 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Suppression subtractive hybrid ...... genes in human mesangial cells
@ast
Suppression subtractive hybrid ...... genes in human mesangial cells
@en
Suppression subtractive hybrid ...... genes in human mesangial cells
@nl
type
label
Suppression subtractive hybrid ...... genes in human mesangial cells
@ast
Suppression subtractive hybrid ...... genes in human mesangial cells
@en
Suppression subtractive hybrid ...... genes in human mesangial cells
@nl
prefLabel
Suppression subtractive hybrid ...... genes in human mesangial cells
@ast
Suppression subtractive hybrid ...... genes in human mesangial cells
@en
Suppression subtractive hybrid ...... genes in human mesangial cells
@nl
P2093
P3181
P356
P1476
Suppression subtractive hybrid ...... genes in human mesangial cells
@en
P2093
H S Mackenzie
P304
P3181
P356
10.1074/JBC.274.9.5830
P407
P577
1999-02-26T00:00:00Z